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Adolescent Problems Treatment
Limping Child Treatment
Management of New Born Care
Treatment of Newborn Jaundice
Treatment of Thyroid Disease in Children
Thyroid Disorder Treatment
Thyroid Problems Treatment
Adolescent Disorders Treatment
Treatment of Child and Adolescent Problems
Treatment of Childhood Diabetes
Cleft Lip Treatment
Management of Postnatal Care
Child Growth Management
Treatment of Childhood Infections
Management of Childhood Nutrition
Congenital Ear Problem Treatment
Quad Screening Treatment
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I have 1 year old child, he is under weight near about 2 kg from standards ,all other things are normal. Please suggest.
Hi. My daughter is 4 month old but she is unable to respond to her neck? Means she can not straight her neck itself when her head is bended right or left.
The appearance of the milk teeth is one of the most awaited landmarks in a child's life. The first teeth to erupt are usually the lower front teeth during 6 to 8 months of life, and the last milk tooth to fall off is at 12 to 14 years of age. The playful nature of teeth, difficulty to make them maintain good oral hygiene, and the food habits put the children at increased risk of dental disease.
Very often, because they are in place for a shorter duration, parents tend to ignore decay in the milk teeth. But whether it is decay or gum disease or broken teeth, it is important to immediately treat them.
Listed below are some functions that milk teeth play:
- Eating: They may be exerting slightly lesser force than the bigger permanent teeth, but they still play a significant role in chewing and digestion. Children with weak, missing, or decayed molars have poor nutrition and food habits due to their inability to chew food well.
- Esthetics: A child with a good set of teeth and an open smile is loved by all. This adds not just to the beauty of the child but also to his self-worth and self-esteem. These children feel more confident and are more social.
- Speech development: A good set of teeth are essential for the child's speech development. Improper spacing between teeth or lost tooth not replaced can lead to speech issues.
- Space Maintenance: In addition to the above functions, the milk teeth also help to preserve and "maintain" the space that is required for the permanent teeth. As the permanent tooth nears eruption, the milk tooth, gets resorbed, becomes mobile, and finally falls off. In cases where the primary tooth was lost and not replaced, the space may be reduced due to movement of the adjacent and opposing tooth into this space.
Given the above reasons, it is very important to take good care of the primary or milk or deciduous teeth. Some simple things to do would be:
- In the very early stages, before regular dental care can begin, the teeth can be wiped off with a gauze wrapped on the finger.
- By the first year of life, brushing should be introduced along with rinsing after each meal.
- A biannual visit to the dentist for oral prophylaxis with regular cleaning should be started by first year of life.
- If the dentist identifies the child to be prone to decay, fluoride application and/or pit and fissure sealants should be used.
These are sufficient reasons to take care of the primary teeth, which play a very important role. If you wish to discuss about any specific problem, you can consult a dentist.
Hi mam or sir. My 9 months old son is suffering from past 4 days with vomit and loose motion. I have consulted doctor it is little bit decreased but be on crying. please tell me how to stop him crying and why he is crying?
Hello doctor I have been suffering from rubella (german measles) I have had headache, cough and cold, red eyes since 2 days I am 21 years old male I want to know the cases of this disease.
My son is 1 month old. Now a days he is not at all drinking breast milk. Will this affect his health.
I have a son 3 years old and having problem of milk allergy, lack of calcium. He is very weak. Kindly suggest to make him healthy.
I have a baby boy. He is 11 month. Uske left cheeks k niche down me red mole jaisa janm se hi h. Iska upaybataye pls ye kya h isse koi isue bhi h?
My son is 7 years old he is very active but in looking very pale and lean. He is taking food in good. He is 118 cm height and 18.5 kg is it good.
My question is for my baby who is 36 days old. She always stretch her body legs and hands and making sound like she feels pain. When I consult with doc she said that it is gas problem or stomach problem. She also prescribed some drops. But still problems is not resolved. Kindly suggest me what I have to do for her. Any medicine or any food which I have to avoid for her.
Gastritis is the most common silent disease of the gastrointestinal tract, affecting more than half of the world population. It is well known that H.pylori is the chief etiological agent of chronic gastritis, peptic ulcer, gastric adenocarcinoma, malt lymphoma. Helicobacter pylorus was discovered by Warren and Marshal in 1983. H. pylori has some unique characteristics:
It defied its detection by scientists for centuries.
It survives in the stomach, an organ which is devised by the nature to kill all bacteria.
85% of the population hosts this organism asymptomatically.
It persists in the gastric mucosa for decades.
It does not penetrate the gastric mucosa for decades.
It reduces the risk of oesophagitis, Barrett’s esophagus, esophageal adenocarcinoma, in the infected individual.
Gastritis is defined as an inflammatory response of the gastric mucosa to infections or irritants.
In the histology of normal gastric mucosa, inflammatory cells – neutrophils are spare and lymphoid tissue is absent.
ACUTE GASTRITIS is diagnosed endoscopically in the presence of hyperemia, intermucosal hemorrhages, and erosions in the gastric antrum and/or body mucosa.
Erosions are flat, or elevated white based lesions with an erythematous margin, and are frequently seen in the antrum.
Histology shows marked surface epithelial degeneration and heavy infiltration with neutrophils, but it is rarely performed.
CHRONIC GASTRITIS may be classified as chronic active, non-atrophic (superficial), atrophic and pernicious anaemia.
On histology of the gastric mucosa, there is a predominant increase in the chronic inflammatory cells – lymphocytes, plasma cells and an occasional lymphoid follicle may be present.
Presence of numerous neutrophils indicates activity (chronic active gastritis).
The vast majority of chronic gastritis patients are asymptomatic. Non colicky pain in upper abdomen within 15 minutes after ingestion of a spicy meal and absence of pain on delaying or omission of a spicy meal are considered suggestive of chronic gastritis. Heaviness in upper abdomen immediately after a meal is also not an uncommon symptom. With a fiberoptic gastroscope a definite diagnosis of chronic gastritis is easy with biopsy from the body mucosa and the antrum. H.pylori causes chronic gastritis in all subjects. H.Pylori colonizes normal antrum and may extend into the body mucosa causing corpus gastritis. Chronic gastritis due to H.pylori slowly progresses over a few decades from the superficial to atrophic gastritis, intestinal metaplasia, dysplasia and gastric adenocarcinoma.
H. pylori was earlier responsible for more than 80% of chronic gastritis but its prevalence is decreasing in countries with improved sanitation.
H.PYLORI AND PEPTIC ULCER
The patients. with duodenal ulcer may present with dull aching pain in the epigastrium, occurring daily on an empty stomach or at midnight relieved soon after the ingestion of antacid, milk or non-spicy food. Nearly half of the numbers of patients with typical history of duodenal ulcer do not show any ulcer on endoscopy. The popular multi-factorial theory of stress and spices causing duodenal ulcer, died its natural death, with the discovery of H.pylori in 1983.
A major breakthrough in understanding of the etiology of duodenal ulcer was the discovery of H.pylori in the antral mucosal biopsy of humans, on upper gastrodudenal endoscopy- as; H.pylori is present in the antral mucosal biopsy of >90 % of duodenal ulcer patients., following the eradication of H.pylori from the gastric mucosa, annual duodenal ulcer recurrence reduced to less than 10% compared to 80%. Failure to eradicate H. pylori results in a higher recurrence rate of duodenal ulcer. H. pylori infection of the antral mucosa increases the risk of duodenal ulcer by 3-6 folds.
Pt. with benign gastric ulcer does not have any classical pattern of symptoms for a clinical diagnosis. Pt. may complain of dull aching pain in upper abdomen soon after food intake, nusea, heaviness, heamatemesis or symptoms of anemia.
Benign gastric ulcer is rare in Indian population, it may occur with ch.gastritis due to H.pylori or following ingestion of aspirin or NSAID. H. pylori increases the risk of benign gastric ulcer by 3 folds.
Gastric mucosal Biopsy
Gastric secretion: Acid, Pepsin, Intrinsic factor
Co vita B12 excretion test
Fasting serum pepsinogen,serum gastrin
Parietal cell, intrinsic factor, helicobacter pylori antibody
H.pylori detection : invasive ,non invasive methods
THE HOMOEOPATHIC APPROACH
Abdominal pain and inflammation present difficulties in diagnosis for even the most experienced physician. All cases of dynamic diseases, acute or chronic even when resulting from mechanical or psychological injuries, are amenable to homoeopathy. The homoeopathic medicine works quite well in the treatment of an acute abdomen often averting the need for surgery in many of cases. The problem may range from entrapment of gas, to constipation, perforation of the bowel which results in sever inflammation and sepsis which may result in death. Any acute onset of abdominal pain should be considered a medical emergency.
By carefully applying the law of similars, the physician will observe that all cases of curable dynamic disease are curable with homoeopathy. To achieve this, the physician must be thoroughly familiar with the principles of homoeopathy as taught in the ORGANON and must know how to make the use of materia medica.
Repertories are used as essential links between the patient’s symptoms and the vast materia medica.
Clinical guides such as below mentioned, provide a synopsis of the most characteristic symptoms of the leading remedies in a given condition. Their objective is to give assistance only. While using it one has to be aware of two general drawbacks. One, it may fail because of its incompleteness as only leading remedies in given a given condition can be presented, and the symptomatology of each remedy presented is limited to only the leading characteristic symptoms.
In clinical practice the patient will most of the time present some symptoms that can only be found in a more complete materia medica. Second, there is the inevitable temptation to associate remedies with a given disease. The practice of homoeopathy consists of constant individualization. – The more we understand this science the more we individualize. Frequent follow up to monitor the patient’s condition is a must.
Gnawing, hungry faint feeling at the epigastrium
Burning and distension of stomach with palpitation
Tendency to eat far beyond the capacity for digestion
Great appetite, craving for meat, pickles, radish, turnips, coarse food
Flatulence disturbs the heart’s action
Wants to lie down all the time
Pain in stomach always comes on after eating
Sensation as if a hard-boiled egg had lodged in the cardiac end of stomach
Great craving for food at noon and night
Dyspepsia of the aged, after tea or tobacco
Constitution – Pale, lean, emaciated persons.
Symptoms relating to GIT indicating hyperacidity – Burning pains as of an ulcer
Cancer of stomach
Vomits every kind of food
Heartburn and water brash
Concomitants – Profuse salivation
Intense burning thirst
Haemorrhage from bowels
ALSO MANY REMEDIES ARS.ALB. , SULPH, CAL.CARB.ETC