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I am 26 years male, am having lipoma skin disease, day by day its increasing on the body like hand, legs, how to reduce and is there any natural cure please let me know.
I haven't been able to sleep since 2 days and I am having a severe headache, tried multiple home remedies, but didn't work, what to do? Please help me!
How to gain weight and mass. What is the best and safe way. To gain weight. Which is the best supplement.
Hi, I have burning sensation and itching around vagina mouth and around my labia for last couple of days. The place does not remain dry no matter how hard I try. No foul smell or thick discharge. I also get vaginal pimples very often. For vaginal pimples I have been given kenozole-bg which I use only when I have a pimple. Just to mention I also have face acne and body acne for which I have done course of doxypal twice in two years. Currently I am on no medication.
I am a anxiety person. I find it hard to breathe when I am stuck with anxiety. What is the first aid and medication
I am 35 year old male, I have infertility problem due to undecending testicals by birth. Now am having single testicle which was placed at 9 years and another removed at 11. My seimen count is nil. Is there any treatment for me to give birth for child.
Can numerous fibroids and an enlarged uterus cause bladder prolapse? Why does my gynecologist think my bladder bulging into my vaginal canal is a fibroid even after I was sent to a urologist for stress incontinence issues?
Hi I am 27 years old & from last 2 years my head hair getting white now my beard hair also becomes white from few spots. Doctors told me that its just because of genetically I got this diseases from my parents now I am worried what to do and which treatment will good for me those suits to pocket as well. My doctor suggest me to take bio tee plus tablet it will be very helpful if you suggest any medicine which stops my hair getting white.
I have sudden discoloration of my lower legs half way up to my calf. I am diabetic and have high blood pressure. The discoloration seems to be clotted blood in thin lines. What is it?
I am 42 and my weight is 97 kg. My height is 6 ft. I am not able to reduce weight. I walk daily for 45 min. Not eat much. Still not able to reduce. Pls help. Thanks.
Since 5 days. I am experiencing pain in my lower stomach & back pain as when as a burning sensation when I pass urine. I feel nauseous most of time and. I had lost my appetite when it happened on the frst day Plss help me.
Age 60 suffering from early symptoms of Osteoarthritis, knee pain. Whether walking to be avoided. Which exercise is advisable? Height is 170 cms and weight 88kg.
Hi sir, I am 20years old, and I have pimples and some scars on my face. What should I do? please suggest me.
Boerhaave first described the spontaneous rupture of the esophagus in 1724. It typically occurs after forceful emesis. Boerhaave syndrome is a transmural perforation of the esophagus to be distinguished from mallory-weiss syndrome, a nontransmural esophageal tear also associated with vomiting. Because it often is associated with emesis, boerhaave syndrome usually is not truly spontaneous. However, the term is useful for distinguishing it from iatrogenic perforation, which accounts for 85-90% of cases of esophageal rupture.
Diagnosis of boerhaave syndrome can be difficult because often no classic symptoms are present and delays in presentation for medical care are common. Approximately one third of all cases of boerhaave syndrome are clinically atypical. Prompt recognition of this potentially lethal condition is vital to ensure appropriate treatment. Mediastinitis, sepsis, and shock frequently are seen late in the course of illness, which further confuses the diagnostic picture.
See can't-miss gastrointestinal diagnoses, a critical images slideshow, to help diagnose the potentially life-threatening conditions that present with gastrointestinal symptoms.
A reported mortality estimate is approximately 35%, making it the most lethal perforation of the gi tract. The best outcomes are associated with early diagnosis and definitive surgical management within 12 hours of rupture. If intervention is delayed longer than 24 hours, the mortality rate (even with surgical intervention) rises to higher than 50% and to nearly 90% after 48 hours. Left untreated, the mortality rate is close to 100%.
Esophageal rupture in boerhaave syndrome is postulated to be the result of a sudden rise in intraluminal esophageal pressure produced during vomiting, as a result of neuromuscular incoordination causing failure of the cricopharyngeus muscle to relax. The syndrome commonly is associated with overindulgence in food and/or alcohol. The most common anatomical location of the tear in boerhaave syndrome is at the left posterolateral wall of the lower third of the esophagus, 2-3 cm proximal to the gastroesophageal junction, along the longitudinal wall of the esophagus. The second most common site of rupture is in the subdiaphragmatic or upper thoracic area. [1, 2]
Although likely underreported, the incidence of boerhaave syndrome is relatively rare. A 1980 review by kish cited 300 cases in the literature worldwide.  a 1986 summary by bladergroen et al described 127 cases.  of these, 114 were diagnosed antemortem; the others were diagnosed at autopsy. Overall, boerhaave syndrome accounts for 15% of all cases of traumatic rupture or perforation of the esophagus.
Race-, sex-, and age-related demographics
Cases have been reported in all races and on virtually every continent, affecting males more commonly than females, with ratios ranging from 2: 1 to 5: 1.
Boerhaave syndrome is seen most frequently among patients aged 50-70 years. Reports suggest that 80% of all patients are middle-aged men. However, this condiction has also been described in neonates and in persons older than 90 years. Although no clear explanation exists for this, the least susceptible age group appears to be children aged 1-17 years.
Prognosis is directly contingent on early recognition and appropriate intervention. Early diagnosis of boerhaave syndrome allows prompt surgical repair. Diagnosis and surgery within 24 hours carry a 75% survival rate. This drops to approximately 50% after a 24-hour delay and approximately 10% after 48 hours.
The mortality rate is high. Esophageal perforation is the most lethal perforation of the gi tract. Survival is contingent largely upon early recognition and appropriate surgical intervention.
Overall, the mortality rate is approximately 30%. Mortality is usually due to subsequent infection, including mediastinitis, pneumonitis, pericarditis, or empyema.
Patients who undergo surgical repair within 24 hours of injury have a 70-75% chance of survival. This falls to 35-50% if surgery is delayed longer than 24 hours and to approximately 10% if delayed longer than 48 hours.
Cases of patients surviving without surgery exist but are rare enough to warrant case reports in the medical literature.
Esophageal rupture may lead to the development of septicemia, pneumomediastinum, mediastinitis, massive pleural effusion, empyema, pneumomediastinum, or subcutaneous emphysema.
If the esophageal rupture extends directly into the pleura, hydropneumothorax is expected. In adults, this occurs more commonly on the left side of the pleura. In neonates, esophageal rupture usually occurs on the right side.
After esophageal rupture, free air enters the mediastinum and also may spread to the adjacent structures, resulting in mediastinal abscess or superimposed secondary infection.
Other complications include acute respiratory distress syndrome, pneumomediastinum, pneumothorax, and hydrothorax.