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Diagnostic Cardiac Procedures
Treatment of Endoscopic Sinus Surgery
Treatment of Lumbar Radiculopathy
Oxygen Therapy Treatment
Hiv Prophylaxis Post Exposure
Restylane Vital Procedure
Treatment of Shin Splints
Treatment of Shin Splits
Management of Smoking Cessation
Treatment of Tetracycline Stains
Weight Management Treatment
Asthma Management Program
Skin Detoxification Treatment
Head And Neck Pain Treatment
Health Check Up
Health Screening For Men
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My penis got whit spot n become skin rashes in circle ,I have faced this problem last few weeks, is this normal?
If a man have to improve his sexual power even he is impotence home remedies any ayurveda doctor plz?
Gastritis is the most common silent disease of the gastrointestinal tract, affecting more than half of the world population. It is well known that H.pylori is the chief etiological agent of chronic gastritis, peptic ulcer, gastric adenocarcinoma, malt lymphoma. Helicobacter pylorus was discovered by Warren and Marshal in 1983. H. pylori has some unique characteristics:
It defied its detection by scientists for centuries.
It survives in the stomach, an organ which is devised by the nature to kill all bacteria.
85% of the population hosts this organism asymptomatically.
It persists in the gastric mucosa for decades.
It does not penetrate the gastric mucosa for decades.
It reduces the risk of oesophagitis, Barrett’s esophagus, esophageal adenocarcinoma, in the infected individual.
Gastritis is defined as an inflammatory response of the gastric mucosa to infections or irritants.
In the histology of normal gastric mucosa, inflammatory cells – neutrophils are spare and lymphoid tissue is absent.
ACUTE GASTRITIS is diagnosed endoscopically in the presence of hyperemia, intermucosal hemorrhages, and erosions in the gastric antrum and/or body mucosa.
Erosions are flat, or elevated white based lesions with an erythematous margin, and are frequently seen in the antrum.
Histology shows marked surface epithelial degeneration and heavy infiltration with neutrophils, but it is rarely performed.
CHRONIC GASTRITIS may be classified as chronic active, non-atrophic (superficial), atrophic and pernicious anaemia.
On histology of the gastric mucosa, there is a predominant increase in the chronic inflammatory cells – lymphocytes, plasma cells and an occasional lymphoid follicle may be present.
Presence of numerous neutrophils indicates activity (chronic active gastritis).
The vast majority of chronic gastritis patients are asymptomatic. Non colicky pain in upper abdomen within 15 minutes after ingestion of a spicy meal and absence of pain on delaying or omission of a spicy meal are considered suggestive of chronic gastritis. Heaviness in upper abdomen immediately after a meal is also not an uncommon symptom. With a fiberoptic gastroscope a definite diagnosis of chronic gastritis is easy with biopsy from the body mucosa and the antrum. H.pylori causes chronic gastritis in all subjects. H.Pylori colonizes normal antrum and may extend into the body mucosa causing corpus gastritis. Chronic gastritis due to H.pylori slowly progresses over a few decades from the superficial to atrophic gastritis, intestinal metaplasia, dysplasia and gastric adenocarcinoma.
H. pylori was earlier responsible for more than 80% of chronic gastritis but its prevalence is decreasing in countries with improved sanitation.
H.PYLORI AND PEPTIC ULCER
The patients. with duodenal ulcer may present with dull aching pain in the epigastrium, occurring daily on an empty stomach or at midnight relieved soon after the ingestion of antacid, milk or non-spicy food. Nearly half of the numbers of patients with typical history of duodenal ulcer do not show any ulcer on endoscopy. The popular multi-factorial theory of stress and spices causing duodenal ulcer, died its natural death, with the discovery of H.pylori in 1983.
A major breakthrough in understanding of the etiology of duodenal ulcer was the discovery of H.pylori in the antral mucosal biopsy of humans, on upper gastrodudenal endoscopy- as; H.pylori is present in the antral mucosal biopsy of >90 % of duodenal ulcer patients., following the eradication of H.pylori from the gastric mucosa, annual duodenal ulcer recurrence reduced to less than 10% compared to 80%. Failure to eradicate H. pylori results in a higher recurrence rate of duodenal ulcer. H. pylori infection of the antral mucosa increases the risk of duodenal ulcer by 3-6 folds.
Pt. with benign gastric ulcer does not have any classical pattern of symptoms for a clinical diagnosis. Pt. may complain of dull aching pain in upper abdomen soon after food intake, nusea, heaviness, heamatemesis or symptoms of anemia.
Benign gastric ulcer is rare in Indian population, it may occur with ch.gastritis due to H.pylori or following ingestion of aspirin or NSAID. H. pylori increases the risk of benign gastric ulcer by 3 folds.
Gastric mucosal Biopsy
Gastric secretion: Acid, Pepsin, Intrinsic factor
Co vita B12 excretion test
Fasting serum pepsinogen,serum gastrin
Parietal cell, intrinsic factor, helicobacter pylori antibody
H.pylori detection : invasive ,non invasive methods
THE HOMOEOPATHIC APPROACH
Abdominal pain and inflammation present difficulties in diagnosis for even the most experienced physician. All cases of dynamic diseases, acute or chronic even when resulting from mechanical or psychological injuries, are amenable to homoeopathy. The homoeopathic medicine works quite well in the treatment of an acute abdomen often averting the need for surgery in many of cases. The problem may range from entrapment of gas, to constipation, perforation of the bowel which results in sever inflammation and sepsis which may result in death. Any acute onset of abdominal pain should be considered a medical emergency.
By carefully applying the law of similars, the physician will observe that all cases of curable dynamic disease are curable with homoeopathy. To achieve this, the physician must be thoroughly familiar with the principles of homoeopathy as taught in the ORGANON and must know how to make the use of materia medica.
Repertories are used as essential links between the patient’s symptoms and the vast materia medica.
Clinical guides such as below mentioned, provide a synopsis of the most characteristic symptoms of the leading remedies in a given condition. Their objective is to give assistance only. While using it one has to be aware of two general drawbacks. One, it may fail because of its incompleteness as only leading remedies in given a given condition can be presented, and the symptomatology of each remedy presented is limited to only the leading characteristic symptoms.
In clinical practice the patient will most of the time present some symptoms that can only be found in a more complete materia medica. Second, there is the inevitable temptation to associate remedies with a given disease. The practice of homoeopathy consists of constant individualization. – The more we understand this science the more we individualize. Frequent follow up to monitor the patient’s condition is a must.
Gnawing, hungry faint feeling at the epigastrium
Burning and distension of stomach with palpitation
Tendency to eat far beyond the capacity for digestion
Great appetite, craving for meat, pickles, radish, turnips, coarse food
Flatulence disturbs the heart’s action
Wants to lie down all the time
Pain in stomach always comes on after eating
Sensation as if a hard-boiled egg had lodged in the cardiac end of stomach
Great craving for food at noon and night
Dyspepsia of the aged, after tea or tobacco
Constitution – Pale, lean, emaciated persons.
Symptoms relating to GIT indicating hyperacidity – Burning pains as of an ulcer
Cancer of stomach
Vomits every kind of food
Heartburn and water brash
Concomitants – Profuse salivation
Intense burning thirst
Haemorrhage from bowels
ALSO MANY REMEDIES ARS.ALB. , SULPH, CAL.CARB.ETC
How to use viagra? What to eat after viagra tab? When to have sex after eating it? What are the side effects caused by his if we use continuously?Please help sir .
Is brisk walking in the evening as good as in the morning. I am not able to go for walking in the morning as I get a up at about 6.45 am and by then it is hot out there. I normally take brisk walk in the evening for 50-55 minutes and am aged 65 years. I am diabetic which is under control at present. Further can I also run though I get pain in my knees while running and get tired soon. I also do some push ups and sit ups in the morning besides a few more exercises. Are these fine for a person my age?
How should the aids is caused Is caused by sex with girl or daily sperm vomiting and how to know the person is caused by aids.
I have lack of blood in my body what things will I take to increase the amount of blood in my body and which things wiil not take.
My aunt has a depression problem. Consulted by a doctor says that its because of pcos. What precautions she should take while going through this illness.
1.3 new goals – cure, prevent resistance and break chain of transmission.
2. Introduction of daily regime.
3. Definition and treatment of mono and polyresistance apart from mdr and xdr tuberculosis.
4. Treatment in cat 1 – 2 (hrze) + 4 (hre): continue ethambutol in continuation phase too.
5. Treatment in cat 2 – 2 (hrzes) + 1 (hrze) + 5 (hre).
6. Introduction of bedaquiline as a new drug. Atp synthase inhibitor specifically targets myc. Tb. Indicated in age more than 18 years. Contraindicatef in pregnancy and those taking hormonal ocp. It may be given in patients with stable arrythmia.
7. Definition of presumptive tuberculosis. Duration > 2 weeks etc.
8. New algorithm to diagnose tuberculosis – pulmonary, extrapulmonary, drug resistant.
9. Introduction of newer molecular methods like cbnaat and line probe assay in diagnostic algorithm apart from smear microscopy and chest xray.
10. Diagnosis of tuberculosis based on x-ray will be called as clinically diagnosed tuberculosis.
11. Sputum should be around 2ml and preferably be mucopurulent.
12. Follow up – new and previously treated drug sensitive pulmonary tuberculosis – no need to extend intensive phase, sputum microscopy at end of ip and end of treatment, weight monthly, chest x-ray if required.
13. Follow up – mdr tuberculosis – sputum smear monthly 3, 4, 5, 6, 7 months in intensive phase and at 3 months interval in continuation phase 9, 12, 15, extend ip phase by maximum 3 months total of 9 months.
Some more additions to it, adding here which might help to pg students.
1) monitoring health status of tb treated patients (for recurrence of tb) for 24 months after treatment
2) online monitoring of treatment adherence through 99dots programme (currently it is on pilot basis running for tb-hiv patients)
3) intensified tb case finding in clinically, socially and geographically vulnerable population. It's a provider initiated activity.
4) now'tb suspect' term is replaced by'presumptive tb case.
5) in diagnostic algorithm sputum examination along with chest x-ray is recommended.
6)'nsp' term is replaced by'microbiologically confirmed case'
7) nsn and others r called now onwards'clinically diagnosed tb' case. (terms replaced)
8) definitions of cured, defaulted, treatment completed, failure, failure to respond, loss to follow up are somwhat changed.
9) cat i, cat ii, cat iv terminologies r obsolete n replaced by drug sensitive (new or previously treated) and drug resistant tb categories.