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Adolescent Problems Treatment
Limping Child Treatment
Management of New Born Care
Treatment of Newborn Jaundice
Treatment of Thyroid Disease in Children
Thyroid Disorder Treatment
Thyroid Problems Treatment
Adolescent Disorders Treatment
Treatment of Child and Adolescent Problems
Treatment of Childhood Diabetes
Cleft Lip Treatment
Management of Postnatal Care
Child Growth Management
Treatment of Childhood Infections
Management of Childhood Nutrition
Congenital Ear Problem Treatment
Quad Screening Treatment
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My son age 3 years what powder should be good for adding of milk is it junior hairloss or prediassuure.
My daughter is 2 years old girl. She eats idli, 2eggs, apple juice, moosambi juice, vegetable soup, rice with any curry, 2 banana, 3 times pasteurized milk; and some biscuits and chocolates. But she is very thin. List what should included in her food items. She is very thin how to becom fat?
Gastritis is the most common silent disease of the gastrointestinal tract, affecting more than half of the world population. It is well known that H.pylori is the chief etiological agent of chronic gastritis, peptic ulcer, gastric adenocarcinoma, malt lymphoma. Helicobacter pylorus was discovered by Warren and Marshal in 1983. H. pylori has some unique characteristics:
It defied its detection by scientists for centuries.
It survives in the stomach, an organ which is devised by the nature to kill all bacteria.
85% of the population hosts this organism asymptomatically.
It persists in the gastric mucosa for decades.
It does not penetrate the gastric mucosa for decades.
It reduces the risk of oesophagitis, Barrett’s esophagus, esophageal adenocarcinoma, in the infected individual.
Gastritis is defined as an inflammatory response of the gastric mucosa to infections or irritants.
In the histology of normal gastric mucosa, inflammatory cells – neutrophils are spare and lymphoid tissue is absent.
ACUTE GASTRITIS is diagnosed endoscopically in the presence of hyperemia, intermucosal hemorrhages, and erosions in the gastric antrum and/or body mucosa.
Erosions are flat, or elevated white based lesions with an erythematous margin, and are frequently seen in the antrum.
Histology shows marked surface epithelial degeneration and heavy infiltration with neutrophils, but it is rarely performed.
CHRONIC GASTRITIS may be classified as chronic active, non-atrophic (superficial), atrophic and pernicious anaemia.
On histology of the gastric mucosa, there is a predominant increase in the chronic inflammatory cells – lymphocytes, plasma cells and an occasional lymphoid follicle may be present.
Presence of numerous neutrophils indicates activity (chronic active gastritis).
The vast majority of chronic gastritis patients are asymptomatic. Non colicky pain in upper abdomen within 15 minutes after ingestion of a spicy meal and absence of pain on delaying or omission of a spicy meal are considered suggestive of chronic gastritis. Heaviness in upper abdomen immediately after a meal is also not an uncommon symptom. With a fiberoptic gastroscope a definite diagnosis of chronic gastritis is easy with biopsy from the body mucosa and the antrum. H.pylori causes chronic gastritis in all subjects. H.Pylori colonizes normal antrum and may extend into the body mucosa causing corpus gastritis. Chronic gastritis due to H.pylori slowly progresses over a few decades from the superficial to atrophic gastritis, intestinal metaplasia, dysplasia and gastric adenocarcinoma.
H. pylori was earlier responsible for more than 80% of chronic gastritis but its prevalence is decreasing in countries with improved sanitation.
H.PYLORI AND PEPTIC ULCER
The patients. with duodenal ulcer may present with dull aching pain in the epigastrium, occurring daily on an empty stomach or at midnight relieved soon after the ingestion of antacid, milk or non-spicy food. Nearly half of the numbers of patients with typical history of duodenal ulcer do not show any ulcer on endoscopy. The popular multi-factorial theory of stress and spices causing duodenal ulcer, died its natural death, with the discovery of H.pylori in 1983.
A major breakthrough in understanding of the etiology of duodenal ulcer was the discovery of H.pylori in the antral mucosal biopsy of humans, on upper gastrodudenal endoscopy- as; H.pylori is present in the antral mucosal biopsy of >90 % of duodenal ulcer patients., following the eradication of H.pylori from the gastric mucosa, annual duodenal ulcer recurrence reduced to less than 10% compared to 80%. Failure to eradicate H. pylori results in a higher recurrence rate of duodenal ulcer. H. pylori infection of the antral mucosa increases the risk of duodenal ulcer by 3-6 folds.
Pt. with benign gastric ulcer does not have any classical pattern of symptoms for a clinical diagnosis. Pt. may complain of dull aching pain in upper abdomen soon after food intake, nusea, heaviness, heamatemesis or symptoms of anemia.
Benign gastric ulcer is rare in Indian population, it may occur with ch.gastritis due to H.pylori or following ingestion of aspirin or NSAID. H. pylori increases the risk of benign gastric ulcer by 3 folds.
Gastric mucosal Biopsy
Gastric secretion: Acid, Pepsin, Intrinsic factor
Co vita B12 excretion test
Fasting serum pepsinogen,serum gastrin
Parietal cell, intrinsic factor, helicobacter pylori antibody
H.pylori detection : invasive ,non invasive methods
THE HOMOEOPATHIC APPROACH
Abdominal pain and inflammation present difficulties in diagnosis for even the most experienced physician. All cases of dynamic diseases, acute or chronic even when resulting from mechanical or psychological injuries, are amenable to homoeopathy. The homoeopathic medicine works quite well in the treatment of an acute abdomen often averting the need for surgery in many of cases. The problem may range from entrapment of gas, to constipation, perforation of the bowel which results in sever inflammation and sepsis which may result in death. Any acute onset of abdominal pain should be considered a medical emergency.
By carefully applying the law of similars, the physician will observe that all cases of curable dynamic disease are curable with homoeopathy. To achieve this, the physician must be thoroughly familiar with the principles of homoeopathy as taught in the ORGANON and must know how to make the use of materia medica.
Repertories are used as essential links between the patient’s symptoms and the vast materia medica.
Clinical guides such as below mentioned, provide a synopsis of the most characteristic symptoms of the leading remedies in a given condition. Their objective is to give assistance only. While using it one has to be aware of two general drawbacks. One, it may fail because of its incompleteness as only leading remedies in given a given condition can be presented, and the symptomatology of each remedy presented is limited to only the leading characteristic symptoms.
In clinical practice the patient will most of the time present some symptoms that can only be found in a more complete materia medica. Second, there is the inevitable temptation to associate remedies with a given disease. The practice of homoeopathy consists of constant individualization. – The more we understand this science the more we individualize. Frequent follow up to monitor the patient’s condition is a must.
Gnawing, hungry faint feeling at the epigastrium
Burning and distension of stomach with palpitation
Tendency to eat far beyond the capacity for digestion
Great appetite, craving for meat, pickles, radish, turnips, coarse food
Flatulence disturbs the heart’s action
Wants to lie down all the time
Pain in stomach always comes on after eating
Sensation as if a hard-boiled egg had lodged in the cardiac end of stomach
Great craving for food at noon and night
Dyspepsia of the aged, after tea or tobacco
Constitution – Pale, lean, emaciated persons.
Symptoms relating to GIT indicating hyperacidity – Burning pains as of an ulcer
Cancer of stomach
Vomits every kind of food
Heartburn and water brash
Concomitants – Profuse salivation
Intense burning thirst
Haemorrhage from bowels
ALSO MANY REMEDIES ARS.ALB. , SULPH, CAL.CARB.ETC
My baby daughter is 4 months old. She is underweight. What all can I give her to feed other than mothers milk? Like orange juice grapes juice? Please help.
My son is 3.5 years old, he got stomach pain first. By taking motion test, the result is hookworm. Then after taking scan, he got mesenteric adenitis. He is taking macox zh kid and senasef. How long will it take to cure? What is the cause for this disease? Is hookworm serious? kindly tell what not to do?
Sudden Infant Death Syndrome (SID) refers to the unexplained and sudden death of a seemingly hale and hearty baby. This condition tends to occur when the baby is asleep and that's why it's also known as crib death. Although the reason for the condition is still not known, many experts have attributed the cause of the condition to abnormal development in the parts of the baby's brain that oversees breathing and awakening from sleep. Nonetheless, there are ways that can prevent the problem from occurring and which are:
- Always place your baby on the back to sleep - Sleeping on the back is the safest position that your baby should be in, whenever he or she sleeps. You shouldn't let your child sleep on the sides as he or she can roll onto the stomach, and may hamper the breathing process. You can place your baby on the stomach when he or she is awake.
- Place your baby on a firm surface to sleep sans of any objects - It's best to place your baby on a firm mattress to sleep while avoiding thick and feather padding like a thick comforter. At the same time, objects like toys, stuffed animals or pillows should be removed from the crib as they may get in the way of your child's breathing by pressing on his or her face.
- Make sure your baby doesn't become very hot - For keeping your baby warm during sleep, it's best to opt for sleep clothing or blanket made of light material so that it doesn't make him or her feel very hot. If using a blanket, it should be placed loosely over the baby and one should also remember to not cover the baby's head during sleep.
- Use a pacifier - Research suggests that the use of the pacifier can reduce a baby's chance of dying from SIDS. This is because the pacifier helps in preventing the baby from rolling over onto his or her stomach during sleep. At the same time, it's also believed that the instrument helps in keeping the baby's tongue positioned in a manner that keeps his or her airways open.
- Breastfeed your baby - Breastfeeding your baby for a minimum of 6 months can help in preventing the occurrence of SIDS. Several studies have revealed this beneficial aspect of breastfeeding.
Related Tip: 4 Worst Foods Ever To Feed Your Baby!