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Adolescent Problems Treatment
Limping Child Treatment
Management of New Born Care
Treatment of Newborn Jaundice
Treatment of Thyroid Disease in Children
Thyroid Disorder Treatment
Thyroid Problems Treatment
Adolescent Disorders Treatment
Treatment of Child and Adolescent Problems
Treatment of Childhood Diabetes
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Management of Postnatal Care
Child Growth Management
Treatment of Childhood Infections
Management of Childhood Nutrition
Congenital Ear Problem Treatment
Quad Screening Treatment
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Hello my son 11 months old. Last week he suffered from fever and it reaches to conversion after dat I take my son to hospital nd he admitted 5 days after discharge he recovered from fever but Dr. says you should take extra care up to 6 years. How I take care my baby? Nd is dis dangerous to my son?
My 10 year old son has asthma. He used asthma preventer. What is the effect of using asthma preventer long term? I heard that using this for year might cause bone shrinkage. Is it true? Are there any supplement or vitamin to help this vision?
The problem of early childhood trauma refers to disturbing experiences that take place in children during 0-6 years of age. The traumas that young children experience can be the effects of natural disaster, war or accidents or that of intentional violence like sexual abuse, domestic violence or physical abuse.
What Does this Condition Impact Childhood?
Traumatic events have a significant impact on your child's life and can break his sense of safety. The problem has been linked with a decrease in the size of the brain cortex. This is the part of the brain that controls several complex functions like thinking, consciousness, memory, attention, awareness, and language. The changes that occur as a result can affect your child's IQ as well as his or her ability to control emotions. As a consequence, he or she may harbor feelings of danger and become more afraid or it may manifest in other personality issues which are not very obvious.
How do you identify this condition?
Generally, children who suffer from traumas face problems in regulating their emotions and behaviors.
They may display signs like:
- Fear of new situations
- Being clingy to their parent(s) or somebody close
- Become easily frightened
- Display aggressive and/or impulsive behavior
- Are difficult to comfort
- Display deterioration in behavior and functioning
- Easily forget newly attained developmental skills
- Difficulty in sleeping
Depending on the age during which your child may go through a traumatic event, he or she can exhibit signs relevant to his or her age.
Children aged 0-2 years may also display reactions like poor verbal skills, memory problems, excessive temper, experience nightmares, etc.
On the other hand, children aged 3-6 years can face difficulties in learning as well as develop poor skill and learning disabilities, face problems in social interaction, may be unable to trust people and so on.
If this condition remains unattended or unresolved for longer, it may create personality issues and can deeply impact the individual's relationships, or equations in the society, as they grow up and even after they become adults. In fact, many behavioral problems can be traced back to childhood trauma.
Related Tip: What Really Causes Personality Disorders?
Gastritis is the most common silent disease of the gastrointestinal tract, affecting more than half of the world population. It is well known that H.pylori is the chief etiological agent of chronic gastritis, peptic ulcer, gastric adenocarcinoma, malt lymphoma. Helicobacter pylorus was discovered by Warren and Marshal in 1983. H. pylori has some unique characteristics:
It defied its detection by scientists for centuries.
It survives in the stomach, an organ which is devised by the nature to kill all bacteria.
85% of the population hosts this organism asymptomatically.
It persists in the gastric mucosa for decades.
It does not penetrate the gastric mucosa for decades.
It reduces the risk of oesophagitis, Barrett’s esophagus, esophageal adenocarcinoma, in the infected individual.
Gastritis is defined as an inflammatory response of the gastric mucosa to infections or irritants.
In the histology of normal gastric mucosa, inflammatory cells – neutrophils are spare and lymphoid tissue is absent.
ACUTE GASTRITIS is diagnosed endoscopically in the presence of hyperemia, intermucosal hemorrhages, and erosions in the gastric antrum and/or body mucosa.
Erosions are flat, or elevated white based lesions with an erythematous margin, and are frequently seen in the antrum.
Histology shows marked surface epithelial degeneration and heavy infiltration with neutrophils, but it is rarely performed.
CHRONIC GASTRITIS may be classified as chronic active, non-atrophic (superficial), atrophic and pernicious anaemia.
On histology of the gastric mucosa, there is a predominant increase in the chronic inflammatory cells – lymphocytes, plasma cells and an occasional lymphoid follicle may be present.
Presence of numerous neutrophils indicates activity (chronic active gastritis).
The vast majority of chronic gastritis patients are asymptomatic. Non colicky pain in upper abdomen within 15 minutes after ingestion of a spicy meal and absence of pain on delaying or omission of a spicy meal are considered suggestive of chronic gastritis. Heaviness in upper abdomen immediately after a meal is also not an uncommon symptom. With a fiberoptic gastroscope a definite diagnosis of chronic gastritis is easy with biopsy from the body mucosa and the antrum. H.pylori causes chronic gastritis in all subjects. H.Pylori colonizes normal antrum and may extend into the body mucosa causing corpus gastritis. Chronic gastritis due to H.pylori slowly progresses over a few decades from the superficial to atrophic gastritis, intestinal metaplasia, dysplasia and gastric adenocarcinoma.
H. pylori was earlier responsible for more than 80% of chronic gastritis but its prevalence is decreasing in countries with improved sanitation.
H.PYLORI AND PEPTIC ULCER
The patients. with duodenal ulcer may present with dull aching pain in the epigastrium, occurring daily on an empty stomach or at midnight relieved soon after the ingestion of antacid, milk or non-spicy food. Nearly half of the numbers of patients with typical history of duodenal ulcer do not show any ulcer on endoscopy. The popular multi-factorial theory of stress and spices causing duodenal ulcer, died its natural death, with the discovery of H.pylori in 1983.
A major breakthrough in understanding of the etiology of duodenal ulcer was the discovery of H.pylori in the antral mucosal biopsy of humans, on upper gastrodudenal endoscopy- as; H.pylori is present in the antral mucosal biopsy of >90 % of duodenal ulcer patients., following the eradication of H.pylori from the gastric mucosa, annual duodenal ulcer recurrence reduced to less than 10% compared to 80%. Failure to eradicate H. pylori results in a higher recurrence rate of duodenal ulcer. H. pylori infection of the antral mucosa increases the risk of duodenal ulcer by 3-6 folds.
Pt. with benign gastric ulcer does not have any classical pattern of symptoms for a clinical diagnosis. Pt. may complain of dull aching pain in upper abdomen soon after food intake, nusea, heaviness, heamatemesis or symptoms of anemia.
Benign gastric ulcer is rare in Indian population, it may occur with ch.gastritis due to H.pylori or following ingestion of aspirin or NSAID. H. pylori increases the risk of benign gastric ulcer by 3 folds.
Gastric mucosal Biopsy
Gastric secretion: Acid, Pepsin, Intrinsic factor
Co vita B12 excretion test
Fasting serum pepsinogen,serum gastrin
Parietal cell, intrinsic factor, helicobacter pylori antibody
H.pylori detection : invasive ,non invasive methods
THE HOMOEOPATHIC APPROACH
Abdominal pain and inflammation present difficulties in diagnosis for even the most experienced physician. All cases of dynamic diseases, acute or chronic even when resulting from mechanical or psychological injuries, are amenable to homoeopathy. The homoeopathic medicine works quite well in the treatment of an acute abdomen often averting the need for surgery in many of cases. The problem may range from entrapment of gas, to constipation, perforation of the bowel which results in sever inflammation and sepsis which may result in death. Any acute onset of abdominal pain should be considered a medical emergency.
By carefully applying the law of similars, the physician will observe that all cases of curable dynamic disease are curable with homoeopathy. To achieve this, the physician must be thoroughly familiar with the principles of homoeopathy as taught in the ORGANON and must know how to make the use of materia medica.
Repertories are used as essential links between the patient’s symptoms and the vast materia medica.
Clinical guides such as below mentioned, provide a synopsis of the most characteristic symptoms of the leading remedies in a given condition. Their objective is to give assistance only. While using it one has to be aware of two general drawbacks. One, it may fail because of its incompleteness as only leading remedies in given a given condition can be presented, and the symptomatology of each remedy presented is limited to only the leading characteristic symptoms.
In clinical practice the patient will most of the time present some symptoms that can only be found in a more complete materia medica. Second, there is the inevitable temptation to associate remedies with a given disease. The practice of homoeopathy consists of constant individualization. – The more we understand this science the more we individualize. Frequent follow up to monitor the patient’s condition is a must.
Gnawing, hungry faint feeling at the epigastrium
Burning and distension of stomach with palpitation
Tendency to eat far beyond the capacity for digestion
Great appetite, craving for meat, pickles, radish, turnips, coarse food
Flatulence disturbs the heart’s action
Wants to lie down all the time
Pain in stomach always comes on after eating
Sensation as if a hard-boiled egg had lodged in the cardiac end of stomach
Great craving for food at noon and night
Dyspepsia of the aged, after tea or tobacco
Constitution – Pale, lean, emaciated persons.
Symptoms relating to GIT indicating hyperacidity – Burning pains as of an ulcer
Cancer of stomach
Vomits every kind of food
Heartburn and water brash
Concomitants – Profuse salivation
Intense burning thirst
Haemorrhage from bowels
ALSO MANY REMEDIES ARS.ALB. , SULPH, CAL.CARB.ETC