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Overview

Montemac Plus 10 Mg/10 Mg Tablet

Manufacturer: Macleods Pharmaceuticals Pvt Ltd
Medicine composition: Bambuterol, Montelukast
Prescription vs.OTC: Prescription by Doctor required

Montemac Plus 10 Mg/10 Mg Tablet is a long acting beta-adrenoceptor agonist, a bronchodilator that is prescribed to patients with asthma. It helps open up the air passage; relieve symptoms like breathlessness and coughing; helps in relaxation of muscles.

Montemac Plus 10 Mg/10 Mg Tablet is not advised for pregnant women, breastfeeding mothers, people with an overactive thyroid gland, high blood sugar, high blood pressure, low levels of potassium in blood or any liver problems. In case of pregnancy or if you are trying for it, consult a doctor regarding its use.

Side effects of Montemac Plus 10 Mg/10 Mg Tablet include tremors, dizziness¸ hypersensitivity reactions, palpitations, headache, nausea, cramps, hypokalaemia, hyperglycaemia, myocardial ischaemia, sleep disturbances and even behavioural disturbances.

The medicine is usually taken once a day before bedtime. Your doctor will tell you about the strength of the dosage.

In addition to its intended effect, Montemac Plus 10 Mg/10 Mg Tablet may cause some unwanted effects too. In such cases, you must seek medical attention immediately. This is not an exhaustive list of side effects. Please inform your doctor if you experience any adverse reaction to the medication.
Is It safe with alcohol?
Romilast b 10 mg/5 mg tablet may cause excessive drowsiness and calmness with alcohol.
Are there any pregnancy warnings?
Romilast b 10 mg/5 mg tablet may be unsafe to use during pregnancy.
Animal studies have shown adverse effects on the foetus, however, there are limited human studies. The benefits from use in pregnant women may be acceptable despite the risk. Please consult your doctor.
Are there any breast-feeding warnings?
Romilast b 10 mg/5 mg tablet is probably unsafe to use during breastfeeding. Please consult your doctor.
Is it safe to drive while on this medicine?
Does this affect kidney function?
Caution should be advised in patients with renal impairment.
Does this affect liver function?
There is no data available. Please consult doctor before consuming the drug.
Below is the list of medicines, which have the same composition, strength and form as Montemac Plus 10 Mg/10 Mg Tablet, and hence can be used as its substitute.
Zydus Cadila
Sun Pharmaceutical Industries Ltd
Avis Lifecare Pvt Ltd
Fourrts India Laboratories Pvt Ltd
Franco-Indian Pharmaceuticals Pvt Ltd
Whenever you take more than one medicine, or mix it with certain foods or beverages, you're at risk of a drug interaction.
What are you using Montemac Plus 10 Mg/10 Mg Tablet for?
Asthma
Disclaimer: The information produced here is best of our knowledge and experience and we have tried our best to make it as accurate and up-to-date as possible, but we would like to request that it should not be treated as a substitute for professional advice, diagnosis or treatment.

Lybrate is a medium to provide our audience with the common information on medicines and does not guarantee its accuracy or exhaustiveness. Even if there is no mention of a warning for any drug or combination, it never means that we are claiming that the drug or combination is safe for consumption without any proper consultation with an expert.

Lybrate does not take responsibility for any aspect of medicines or treatments. If you have any doubts about your medication, we strongly recommend you to see a doctor immediately.

Popular Questions & Answers

I always have cold starting from Morning to noon or sometimes full day. If I have allergy medicines its fine. I take montemac fx when I feel colds too much. I took these meds for 2 months, but as soon as they are off. It starts again. Please let me know what further steps should I take.

MBBS, MD TUBERCULOSIS AND CHEST DISEASES, Diploma in Tuberculosis & Chest Diseases, Diploma in Tropical Medicine and Hygiene
Pulmonologist, Kolkata
I always have cold starting from Morning to noon or sometimes full day. If I have allergy medicines its fine. I take ...
You should wear facemask to avoid exposure to dust fumes irritants, if you follow this drug requirement will be less there is no permanent cure.

Can I give ibugesic plus 5 ml Flora bc 2.5 ml & montemac fx 2.5 ml to my 7 years old daughter for cough cold & fever.

MBBS, MD TUBERCULOSIS AND CHEST DISEASES, Diploma in Tuberculosis & Chest Diseases, Diploma in Tropical Medicine and Hygiene
Pulmonologist, Kolkata
Can I give ibugesic plus 5 ml Flora bc 2.5 ml & montemac fx 2.5 ml to my 7 years old daughter for cough cold & fever.
Ibugesic is contraindicated in Asthma or any cough and cold. Paracetamol is a safer option for fever in such conditions. Wish your daughter good health.

I am suffering from nasal polyps for few years. My nose get blocked at night and I have cold like symptoms most of the time. Kindly suggest medicine and drops for its treatment.

Bachelor of Ayurvedic Medicines and Surgery(BAMS), Post Graduation Diploma in Emergency Medicines And Services(PGDEMS), MD - Alternate Medicine
Ayurveda, Ghaziabad
I am suffering from nasal polyps for few years. My nose get blocked at night and I have cold like symptoms most of th...
Take pranacharya allergin capsule 1-1 twice a day. 1-1 drop of cow ghee in both nostrils at night time before sleep. Avoid milk. Cold drinks. Take hot water steam.

Whether long use of Seroflo 250 for asthma is harmful. Which medicine is suggested for very occasional asthma.

PDDM, MHA, MBBS
General Physician, Nashik
Whether long use of Seroflo 250 for asthma is harmful. Which medicine is suggested for very occasional asthma.
Montemac L is a controlled medicine for allergic asthma. If you are able to remain symptom free by using it daily, I would suggest you to continue it for at least 3 months continuously. I would suggest you to consult an Allergist-Immunologist who will suggest you allergy testing. This will help you identify substances causing symptoms to you as well as precautions regarding how to avoid them. Also, an Allergist may suggest you specific allergen immunotherapy depending upon the results of allergy testing, which may improve your allergy symptoms. Please try to avoid exposure to smoke, dusts and air pollution as much as possible. Also please do not smoke if you are a smoker. A nutritive diet rich in vitamins & minerals including anti-oxidants is very important to support immune system. I would also suggest you regular breathing exercises which help you in a long run.
1 person found this helpful

Popular Health Tips

Seasonal Wheezing - Best Ways It Can Be Controlled!

MBBS, MD - Internal Medicine
Internal Medicine Specialist, Faridabad
Seasonal Wheezing - Best Ways It Can Be Controlled!

Seasonal changes can be very exciting for most people. Each season brings with it a new set of colours and nature takes a new turn. However, for people with seasonal allergies, a seasonal change also comes with a set of allergies. From skin rashes to wheezing to breathing difficulties, the list of symptoms can be quite long. Asthma is the most common problem, and the attacks can be quite bothersome with wheezing attacks and breathing troubles.

With advancements in the field of medicine, there is a lot of relief for asthma patients. A little understanding on how asthma attacks happen will help in understanding how to control it. Asthma is an allergic reaction caused by narrowing of the airway with thick mucus, which makes breathing difficult. During an attack, the person can have a dry cough, face shortness of breath and wheezing.

Asthma is often triggered or worsened by some factors, and being aware of these can help prevent or manage an attack:

  1. Common allergens like pollen, mites, ticks, furs of animals, bird feathers, specific foods (peanuts, milk products, eggs, etc.) and mold spores appear during change of season.
  2. Environmental chemicals like cigarette smoke, car fumes, dust, etc., can also trigger an attack.
  3. Attacks of cold or flu can lead to an asthma attack.
  4. Workout during cold weather can be problematic, but exercise can also help control asthma attacks. Lung function improves sufficiently, but an exercise regime should be drawn up for the patient based on his condition.
  5. Stressful emotions like anger and anxiety can also lead to an attack or make it worse.
  6. Some medications like aspirin, beta blockers, glaucoma drops can aggravate attacks of asthma.

Once you know the triggers, here are some common measures that can help prevent and relieve the wheezing.

  1. The preventers reduce the inflammation in the airway tube and the swelling. While they do not provide immediate relief during an attack, using these in the long term helps avoid attacks. These are low-dose inhaled corticosteroids such as beclomethasone, fluticasone, and budesonide.
  2. Doctors would advise their usage even when there are no symptoms, as asthma attacks can be triggered when they are not taking these medications for a while. Newer drugs include leukotriene antagonists like montelukast and zafirlukast.
  3. Relievers are used for symptom relief and include Salbutamol (short acting) and Salmeterol/Formoterol (long acting). Peak flow meter may be useful in acute attacks, wherein the peak flow rates can be reduced.

A good strategy is to use preventers regularly and rely on relievers during an attack. The first one helps build resistance and so reduces the incidence of attacks. Reach out to a doctor if a severe attack ensues (lasts more than 3 hours).

In case you have a concern or query you can always consult an expert & get answers to your questions!

5674 people found this helpful

Parkinson s Disease

MBBS, DNB, Fellowship In Neurosurgery
Neurosurgeon, Kolkata
Parkinson s Disease

Deep brain stimulation in Parkinson’s disease

Abstract: Deep brain stimulation (DBS) is a widely accepted therapy for medically refractory Parkinson’s disease (PD). Both globus pallidus internus (GPi) and subthalamic nucleus (STN) stimulation are safe and effective in improving the symptoms of PD and reducing dyskinesias. STN DBS is the most commonly performed surgery for PD as compared to GPi DBS. Ventral intermediate nucleus (Vim) DBS is infrequently used as an alternative for tremor predominant PD patients.

Patient selection is critical in achieving good outcomes. Differential diagnosis should be emphasized as well as neurological and nonneurological comorbidities. Good response to a levodopa challenge is an important predictor of favorable long-term outcomes. The DBS surgery is typically performed in an awake patient and involves stereotactic frame application, CT/MRI imaging, anatomical targeting, physiological confirmation, and implantation of the DBS lead and pulse generator. Anatomical targeting consists of direct visualization of the target in MR images, formula-derived coordinates based on the anterior and posterior commissures, and reformatted anatomical stereotactic atlases. Physiological verification is achieved most commonly via microelectrode recording followed by implantation of the DBS lead and intraoperative test stimulation to assess benefits and side effects. The various aspects of DBS surgery will be discussed.

Key words: deep brain stimulation (DBS); Parkinson’s disease(PD),  stereotaxis

Introduction

Parkinson's disease is a slowly progressive, neurodegenerative disease characterized by tremor, rigidity, bradykinesia and postural instability. It is the most common movement disorder in middle or late life with a prevalence of about 0.3% of the general population, rising to 1% in people over 60 years of age. Approximately 130 000 people suffer from it in the UK and it presents an increasing burden in our ageing population. Pathological findings in Parkinson's disease demonstrate greatly diminished neuromelanin pigmented neurons in the substantia nigra of the basal ganglia with associated gliosis, and Lewy bodies present in many remaining neurons.

James Parkinson, in his original 1817 Essay on The Shaking Palsy, gave an account of six patients in which he noted signs of tremor, festinating gait and flexed posture.  Nearly two centuries from Parkinson's observations, and almost four decades after Cotzias' dramatic demonstration of levodopa's efficacy, the limitations and complications of levodopa treatment for Parkinson's disease have become well documented Five years after initiation of therapy, a majority of patients develop medication related motor complications, namely levodopa induced dyskinesias (LID) and motor fluctuations. Deep brain stimulation (DBS) has been developed primarily to address these treatment related motor complications and therapeutic failures.

Pathophysiology of PD

The loss of dopaminergic neurons in the substantia nigra, the main functional characteristic of PD, affects the circuit described above and leads to the cardinal motor symptoms of PD. While the exact mechanism of this process is unknown, animal research as well as human recordings have provided functional and biochemical evidence that bradykinesia in PD results from excessive activity in the STN and the GPi. This leads to an exaggerated beta (10-30 Hz) synchronization within and between structures in the basal ganglia circuitry  that could also contribute to rigidity and akinesia.

The pathophysiology of rest tremor in PD is less clear and probably more complicated. This symptom most likely results from a dysfunction of both the striato-pallidal-thalamocortical and the cerebellodentato-thalamocortical circuits, with hyperactivity and hypersynchronization between central oscillators.

Possible mechanism of action of DBS

DBS acts through delivering an electrical current in a specific target area of the brain. This current can be modulated through modification of voltage, frequency and duration of each electrical pulse delivered. The delivered energy creates an electrical field of variable size and shape according to the parameters used for stimulation. Although initially believed to stimulate the target, thus the name of the whole process, it seems that

DBS actually excites the neuronal fibers, but inhibits the neural cells. In fact, GPi DBS decreases the GPi mean firing rate back to a normal range in animal models as well as PD patients, and high frequency DBS has a similar effect as dopamine replacement therapies, and promotes faster (about 70 Hz) nonhypersynchronous activity in the basal ganglia, correlated with clinical improvement. This might be achieved through stimulation of bypassing inhibitory pathways, synaptic inhibition, depolarizing blockade, synaptic depression, and simulation-induced disruption of pathological network activity. Overall, this leads to modifications of the firing rate and pattern of neurons in the basal ganglia, as well as local release of neurotransmitters such as glutamate and adenosine. In addition, it seems that DBS also increases blood flow and stimulates neurogenesis. Over the last few years, functional imaging, specifically functional magnetic resonance imaging (fMRI), positron emission tomography (PET) and single-photon emission computed tomography (SPECT), has been used in an attempt to clarify the mechanism of action of DBS. In fMRI, blood-oxygen-level-dependent (BOLD) signals are acquired, and oxygenated blood marks areas of neural stimulation or inhibition. On the other hand, PET and SPECT allow for imaging of multiple activity markers, such as blood flow, glucose and oxygen metabolism. While fMRI is less powerful than nuclear medicine techniques, it provides a much better spatial and temporal resolution. Because of the suspected inhibitory DBS effects in electrophysiological studies, reduced STN blood flow or glucose metabolism would have been expected on functional imaging. However, the opposite has been found to be true in an overwhelming majority of imaging studies to date. In addition, BOLD activation in the area surrounding the electrode has been reported, despite the electrode imaging artifact preventing direct observation of the STN around the electrode. This discrepancy between apparent STN inhibition in single-cell studies and activation in imaging studies might be explained by a few hypotheses. First, electrophysiological recordings identify short neuronal modulation (in the order of milliseconds) while neuroimaging methods may reflect the summed activity changes over seconds to minutes. Second, non-neuronal contributions to the change in blood flow and/or glucose metabolism cannot be excluded, and could confound the results of neuroimaging.

Finally, it is possible that PET and fMRI actually detect the increased activity in the axons, rather than in the cell bodies. Complicating matters further, some imaging studies after STN DBS have showed increased

activity in the GPi while others reported decreased activity in that nucleus. In summary, it is still unclear how exactly DBS affects the firing rate and pattern of neurons and how these changes actually modify the symptoms of Parkinson’s disease. DBS is presently more of an empirically proven treatment in search of physiological explanation.

The effect of DBS on the cardinal symptoms of PD have been established in three randomized controlled clinical trials --- 

TABLE 1

Author, year

 

No of patients

Follow up

Target

Results

Deuschl et al., 2006

156

6 months

BL STN

QOL better with DBS, motor symptom better with DBS

 

Weaver et al., 2009

255

6 months

BL STN or GPi

Dyskinesia free ON time better with DBS

 

Williams et al., 2010

366

12 months

BL STN  or GPi

QOL better with DBS

 

 

PATIENT SELECTION for DBS in PD

Patient selection is a critical first step as poorly chosen candidates may not have optimal benefits and have increased morbidity. Several factors must be considered before determining if a patient is an appropriate candidate for DBS surgery. A multidisciplinary approach involving the neurosurgeon, neurologist, and neuropsychologist is important to determine the appropriate surgical candidate. It is also important that the diagnosis of idiopathic PD be confirmed prior to proceeding with DBS surgery. Key to this assessment is evaluating the surgical candidate in both the on and off medication states with a corroborating levodopa challenge. Perhaps the best prognostic indicator of a patient’s suitability for DBS surgery is their response to levodopa.In general, a levodopa challenge following a 12-hour medication withdrawal should provide at least a 33% improvement in the motor section of the Unified Parkinson’s Disease Rating Scale (UPDRS).

                     In our institute, we follow a simple chart(below) for screening of patients for DBS in PD.

 

 

  1.  

Age<75 years

 

  •  

No

  1.  

Idiopathic PD ( No PSP/MSA/NSD etc)

 

  •  

No

  1.  

Levodopa responsive  

                      

  •  

No

  1.  

Poor/adverse response to drug          

 

  1.  Increased off period                                                              

 

  1. Disabling dyskinesia                                                              

 

 

  1. Disabling motor fluctuations                 

 

 

Yes

 

Yes

 

 

Yes

 

 

No

 

No

 

 

No

  1.  

Degree of disability(UPDRS part III score)>25

 

  •  

No

  1.  

Neuropsychology, MMSE>24

 

  •  

No

  1.  

LEVODOPA CHALLENGE RESPONSE POSITIVE                                                   

 

(30% improvement in UPDRS after 12-hours off medication)

 

  •  

No

  1.  

Advanced  co-morbidity

 

Yes

  •  
  1.  

long term anticoagulation

 

Yes

  •  
  1.  

Willing for surgery and programming

 

  •  

No

 

 

PREOPERATIVE MANAGEMENT

A full medical assessment is a necessary part of the preoperative evaluation, as advanced PD patients tend to be elderly with significant comorbidities. Major issues are---

 

Anticoagulation/antiplatelets--- The risk of discontinuing medications that affect anticoagulation and

platelet aggregation should be weighed against the potential benefits in the quality of life offered by DBS surgery. However, timely discontinuation of these latter medications is mandatory for stereotactic surgery since intracerebral hematomas are the most serious of all potential complications from DBS. Any anticlotting medications, including aspirin, ticlopidine, clopidogrel, and all nonsteroidal anti-inflammatory drugs should be discontinued at least 7 to 10 days preoperatively to ensure the return of normal blood clotting function.

Arterial hypertension can also increase the risk of intracranial bleeding during stereotactic procedures and must be controlled in the weeks prior to surgery.

A prolonged discussion on the short- and long-term effects of DBS on Parkinson’s disease should be carried out with the patient, family, and caregivers.

The night prior to DBS surgery, the antiparkinsonian medications are typically held to pronounce the Parkinson’s symptoms at the time of surgery to see the clinical effects on symptoms during surgery and the families must be counselled regarding their role in facilitating the patient.

Target selection

The two main targets considered for DBS in PD are the STN and the GPi. current tendency is to prefer targeting the STN because of a greater improvement in the OFF phase motor symptoms as well as a higher chance to decrease the medication dosage and a lower battery consumption linked to the use of lower voltage in the STN compared to the GPi DBS. GPi can be the preferred target if LID is the main complaint. GPi DBS might be preferred for patients with mild cognitive impairment and psychiatric symptoms. Because STN DBS might have a higher rate of cognitive decline and/or depression and worsening of verbal fluency in some studies.

Surgical technique

The basic components of DBS implantation surgery involve frame placement, anatomical targeting, physiological mapping, evaluation of macrostimulation thresholds for improvement in motor symptoms or induction of side effects, implantation of the DBS electrode and implantable pulse generator (IPG).

Head-frame placement

The CRW frame is the most commonly used followed by the Leksell frame. Placement of the frame is done under local anesthesia unless anxiety or uncontrollable movements necessitate the use of sedation or general anesthesia.

Leksell stereotactic frame  placed over the head of a patient showing the correct method for placement of the Leksell head-frame. The frame should be placed parallel to orbito-meatal line in order to approximate the AC-PC plane. It is attached to the patient’s head using four pins under local anesthesia.

Imaging and anatomic targeting

Computerized Tomography (CT) scans and MRI are the two main imaging modalities used for targeting when performing DBS implantations. A thin cut stereotactic CT (_2 mm slices with no gap and no gantry tilt) is obtained after frame placement and is then fused with the stereotactic MRI on a planning station (Stealth station). The advantage of fusing the CT with MRI is the ability to avoid image-distortions inherent to MR imaging adding to the stereotactic accuracy. To better define the STN, T2-weighted images (TR 2800, TE 90, flip angle 90˚, slice thickness 2.0 mm) were obtained.

The AC and the PC were marked and the centre of the AC–PC line determined. The next step is planning the entry point and trajectory. The strategy here is to avoid surface and sub-cortical vessels. After trajectory planning, the patient is placed supine on the operating table and the frame attached to the table using an adaptor. Prophylactic antibiotics are given at least 30 min prior to incision. The head is prepped and draped in a sterile fashion. Under local anesthesia, a burr-hole is placed on the calculated entry point marked on the skull. The entry point is determined by the calculated arc and ring angles. Hemostasis is achieved with bone wax and bipolar cautery.

A Medronic Stim-Loc anchoring device (Medtronic, Minneapolis, MN) burr-hole base ring is then placed on the burr-hole and secured with two screws which are used at the end of the procedure to anchor the DBS electrode.

The dura is then cauterized and opened exposing the underlying surface of the brain. The microdrive is then assembled and cannulae inserted 10 mm above the target to avoid lenticulostriate vessels found deeper. Gel- foam and fibrin glue is applied on dural hole to minimize cerebrospinal fluid (CSF) loss and air entry into the skull. Subsequently, microelectrode recording and stimulation is undertaken.

Microelectrode recording/ Mapping

Microelectrode mapping is used to precisely define the target STN and its boundaries as well as nearby critical structures. We believe microelectrode mapping is crucial in order to give one the best chance for optimal placement of the DBS lead given anatomical inaccuracies due to image distortion and intraoperative brain shifts secondary to CSF loss, and pneumocephalus that can lead to inaccuracies in defining the initial target coordinates and shifts in the target itself once the skull is opened. Microelectrode mapping is performed using platinum-iridium glass coated microelectrodes dipped in platinum black with an impedance of around 0.3–0.5 Mo. These platinum-iridium microelectrodes are capable of recording single unit activity and can also be used for micro-stimulation up to 100 mAwithout significant breakdown in their recording qualities.

As the recording electrode was advanced, entry into the STN was identified by a sudden increase in the density of cellular discharge, with the characteristic irregular pattern of discharge—spikes of different sizes, occurring at random intervals. On coming out of the STN a quiet period (background noise) was seen followed by recording from the substantia nigra if the recording was continued far enough, described as high frequency (50–60 spikes/s) discharge pattern.11 Characteristic STN recordings (visual and audio) were identified and the depth of the STN activity was noted. Identification of STN activity was only based on the visual identification. The centre of the point of best electrical activity was selected as the final target. The microelectrode was replaced with a permanent quadripolar macroelectrode (Medtronic electrode no. 3389) to target the centre of the STN electrical activity. The proximal part of this electrode consists of four nickel conductor wires insulated with a polytetrafluoroethylene jacket tubing. The distal part has four metallic noninsulated contacts of 1.5 mm spaced at 0.5 mm intervals. The diameter of the distal electrode is 1.27 mm. Based on the clinical response any of the four contacts can be used for stimulation. Macrostimulation using the DBS electrode itself is then used to determine benefits and side effects. In most cases lateral skull x rays were obtained at this point with image intensifier carefully positioned to locate the target point in the centre of the Leksell-G frame rings.

Initial programming is always refined by using intra-operative macrostimulation data and a mono-polar review to identify the thresholds of stimulation for improvement in parkinsonian motor signs as well as the thresholds for inducing side effects at the level of each contact. The four variables that are used in programming are choice of contacts (0, 1, 2 or 3 used either as the cathode or anode), frequency of stimulation (hertz), pulse-width (ms) and amplitude (voltage).

POSTOPERATIVE MANAGEMENT

In the immediate hours after surgery, it is important to keep arterial blood pressure in the normal range. In addition, the patient’s preoperative drug regimen should be restarted immediately after surgery to avoid problems with dopaminergic withdrawal. Patients should undergo postoperative CT scans and/or MRI scans to assess the electrode location and intracranial status. In addition, plain X-rays are obtained to assess the location and geometry of the leads and hardware. Parkinson’s medications may need to be adjusted depending on the patient’s status. Cognitive and behavioral changes may occur in the postoperative period, particularly in older patients. Patients can be discharged as early as 24 hours after surgery, depending on their neurological and cognitive status.

Conclusion

For the last 50 years, levodopa has been the cornerstone of PD management. However, a majority of patients develop motor fluctuations and/or LID about 5 years after the initiation of therapy. DBS of the STN or the GPI grant to patients with PD improved quality of life and decreased motor complications, and has been approved as such by the Food and Drug Administration in the US in 2002. We reviewed the experience and available literature on DBS for Parkinson’s disease over the last decade and arrive at the following understandings.

The success of DBS surgery depends on the accurate placement of the leads and meticulous programming of the stimulation. Therefore, it is best accomplished by an experienced team of neurosurgeon, neurologist, and support staff dedicated to the treatment.

Reports of surgical complication rates and long-term side-effects of DBS are very variable, so benefits and potential adverse results should not be under- or over-emphasized.

While essentially equal in improving the motor symptoms of PD, STN and GPi might have their own benefits and risks, and the choice of the target should be individualized and adapted to the patient’s situation.

Knowledge to further improve DBS treatment for Parkinson’s disease, such as a more scientific and reliable protocol on programming, strategies to minimize cognitive and psychiatric complications, and the better

long-term maintenance of the implanted device, are still lacking.

Data on the impact of DBS on non-motor symptoms affecting the quality of life of PD patients, such as pain, speech or gastro-intestinal complaints, are still scarce. Further research in these areas will help make this useful treatment even more beneficial.

4 people found this helpful

Tips to Control Allergy Driven Wheezing this Season

MBBS, DNB (General Medicine), Certified in Evidence Based Diabetes Management, MNAMS, MRCP (UK)
Internal Medicine Specialist, Kolkata
Tips to Control Allergy Driven Wheezing this Season

Seasonal changes can be very exciting for most people. Each season brings with it a new set of colours and nature takes a new turn. However, for people with seasonal allergies, a seasonal change also comes with a set of allergies. From skin rashes to wheezing to breathing difficulties, the list of symptoms can be quite long. Asthma is the most common problem, and the attacks can be quite bothersome with wheezing attacks and breathing troubles.

With advancements in the field of medicine, there is a lot of relief for asthma patients. A little understanding on how asthma attacks happen will help in understanding how to control it. Asthma is an allergic reaction caused by narrowing of the airway with thick mucus, which makes breathing difficult. During an attack, the person can have a dry cough, face shortness of breath and wheezing.

Asthma is often triggered or worsened by some factors, and being aware of these can help prevent or manage an attack:

  1. Common allergens like pollen, mites, ticks, furs of animals, bird feathers, specific foods (peanuts, milk products, eggs, etc.) and mold spores appear during change of season.
  2. Environmental chemicals like cigarette smoke, car fumes, dust, etc., can also trigger an attack.
  3. Attacks of cold or flu can lead to an asthma attack.
  4. Workout during cold weather can be problematic, but exercise can also help control asthma attacks. Lung function improves sufficiently, but an exercise regime should be drawn up for the patient based on his condition.
  5. Stressful emotions like anger and anxiety can also lead to an attack or make it worse.
  6. Some medications like aspirin, beta blockers, glaucoma drops can aggravate attacks of asthma.

Once you know the triggers, here are some common measures that can help prevent and relieve the wheezing.

  1. The preventers reduce the inflammation in the airway tube and the swelling. While they do not provide immediate relief during an attack, using these in the long term helps avoid attacks. These are low-dose inhaled corticosteroids such as beclomethasone, fluticasone, and budesonide. Doctors would advise their usage even when there are no symptoms, as asthma attacks can be triggered when they are not taking these medications for a while. Newer drugs include leukotriene antagonists like montelukast and zafirlukast. Relievers are used for symptom relief and include Salbutamol (short acting) and Salmeterol/Formoterol (long acting). Peak flow meter may be useful in acute attacks, wherein the peak flow rates can be reduced.

A good strategy is to use preventers regularly and rely on relievers during an attack. The first one helps build resistance and so reduces the incidence of attacks. Reach out to a doctor if a severe attack ensues (lasts more than 3 hours).
 

3416 people found this helpful

Kidney Transplant

DNB (Urology), MS - General Surgery, MBBS
Urologist, Delhi
Play video

Here are basic tips of Kidney Transplantation (Renal transplantation)

Friends good after noon I am DR. Aditya Pradhan. I look after the department of urology and kidney transplant at the BL Kapoor hospital. In my department major activities are kidney transplant, kidney cancers we do the constructive urology we also do a lot of stone surgery and prostate surgery so literally, we cover the whole spectrum of urology practice as it is done today in most centers around the world to speak briefly about kidney transplant which is the focus of our activity so as you must be aware India has a large number of patients with kidney failure on an average in India about to lac patients are diagnosed with kidney failure every year. Unfortunately out of these two lac patients barely 3,000 patients get your kidney transplant done every year so there is a large number of patients who don’t get a kidney transplant done in spite of the facilities being available in most major cities in the country.

There are many reasons why a kidney transplant is not easily available- 

The first and most forms, of course, is the availability of donor so there are stringent laws governing the rules of a kidney transplant and it is mandatory for every patient to get his own donor and these donors have to be live related which means first-degree blood relatives. They could be the patient’s parents they could be a sibling, could be his own children who are above 18 years of age or they could be his spouse so amongst these family members, he has to get a patient of the same blood group or of O blood group. Many families do not have relatives with these conditions and that is why there is a big problem for the patient to get a donor for him.  So, this is one of the main problems that you have, that you don’t have an adequate number of donors.

The second problem is even when donors are available within the family they are not willing to donate their kidneys. There are some reasons which are their most of them are misconceptions that patient was donated his kidney may not be able to function normally after the operation. So, these are absolutely unfounded doubts every patient who is going to be tested for kidney donation. All his tests are done to confirm that he is healthy the operation is usually a very safe operation and after the surgery, the patient can lead absolutely normal life.

These misconceptions must be discarded by the donor. They must come to the transplant surgeon and once they counseled and I am pretty sure that they will understand that they are doing a life-saving deed for their near and dear ones. This is the second problem where donors are available but they are not willing to help their relatives because of this misconception about the operation now the third reason is that we don’t have many cadaver donations.

In India now some of you may be aware that there is a concept called brain stem death these are patients who died in the ICU. Before their patients are declared dead their relatives are counseled that they can help the community by donating organs. These are what other problems which are faced when we are doing kidney transplants today but the scenario in India is changing, we have become increasingly better in our techniques we have got good drugs available so in most instances, a kidney transplant is a successful operation in this hospital.

Our results are more than ninety-nine percent which is as good as world standards we are doing all kinds of transplants the kidney donor operation between laparoscopic ally so that the recovery is very quick and most donors can be discharged by the third day to go back home. we are also doing abo incompatible transplant which means that if the blood groups are not matching the donor can be still be taken up for the transplant.

so friends this was a brief about kidney transplant and if you would like any more details about any of the aspects which I talked about you can reach out to me at the lybrate.com website.
 

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Avoid The Ban Medicines To Be Safe

BHMS
Homeopath, Singrauli
Avoid The Ban Medicines To Be Safe

Govt bans 344 drugs, including phensedyl, corex
Sr. No. Product name (irrational fdc) 
1 aceclofenac + paracetamol + rabeprazole
2 nimesulide + diclofenac
3 nimesulide + cetirizine + caffeine
4 nimesulide + tizanidine
5 paracetamol + cetirizine + caffeine
6 diclofenac + tramadol + chlorzoxazone
7 dicyclomine + paracetamol + domperidone
8 nimesulide + paracetamol
9 paracetamol + phenylephrine + caffeine
10 diclofenac+ tramadol + paracetamol
11 diclofenac + paracetamol + chlorzoxazone + famotidine
12 naproxen + paracetamol
13 nimesulide + serratiopeptidase
14 paracetamol + diclofenac + famotidine
15 nimesulide + pifofenone + fenpiverinium + benzyl alcohol
16 omeprazole + paracetamol + diclofenac
17 nimesulide + paracetamol injection
18 tamsulosin + diclofenac
19 paracetamol + phenylephrine + chlorpheniramine + dextromethorphan + caffeine
20 diclofenac + zinc carnosine
21 diclofenac + paracetamol + chlorpheniramine maleate + magnesium trisillicate
22 paracetamol + pseudoephedrine + cetrizine
23 phenylbutazone + sodium salicylate
24 lornoxicam + paracetamol + trypsin
25 paracetamol + mefenamic acid + ranitidine + dicylomine
26 nimesulide + dicyclomine
27 heparin + diclofenac
28 glucosamine + methyl sulfonyl methane + vitamini d3 + maganese + boron + copper + zinc
29 paracetamol + tapentadol
30 tranexamic acid + proanthocyanidin
31 benzoxonium chloride + lidocaine
32 lornoxicam + paracetamol + tramadol
33 lornoxicam + paracetamol + serratiopeptidase
34 diclofenac + paracetamol + magnesium trisilicate
35 paracetamol + domperidone + caffeine
36 ammonium chloride + sodium citrate + chlorpheniramine maleate + menthol
37 paracetamol + prochlorperazine maleate
38 serratiopeptidase (enteric coated 20000 units) + diclofenac potassium &amp; 2 tablets of doxycycline
39 nimesulide + paracetamol suspension
40 aceclofenac + paracetamol + famotidine
41 aceclofenac + zinc carnosine
42 paracetamol + disodium hydrogen citrate + caffeine
43 paracetamol + dl methionine
44 disodium hydrogen citrate + paracetamol
45 paracetamol + caffeine + codeine
46 aceclofenac (sr) + paracetamol
47 diclofenac + paracetamol injection
48 azithromycin + cefixime
49 amoxicillin + dicloxacillin
50 amoxicillin 250 mg + potassium clavulanate diluted 62.5 mg
51 azithromycin + levofloxacin
52 cefixime + linezolid
53 amoxicillin + cefixime + potassium clavulanic acid
54 ofloxacin + nitazoxanide
55 cefpodoxime proxetil + levofloxacin
56 azithromycin, secnidazole and fluconazole kit
57 levofloxacin + ornidazole + alpha tocopherol acetate
58 nimorazole + ofloxacin
59 azithromycin + ofloxacin
60 amoxycillin + tinidazole
61 doxycycline + serratiopeptidase
62 cefixime + levofloxacin
63 ofloxacin + metronidazole + zinc acetate
64 diphenoxylate + atropine + furazolidonee
65 fluconazole tablet, azithromycin tablet and ornidazole tablets
66 ciprofloxacin + phenazopyridine
67 amoxycillin + dicloxacillin + serratiopeptidase
68 azithromycin + cefpodoxime
69 lignocaine + clotrimazole + ofloxacin + beclomethasone
70 cefuroxime + linezolid
71 ofloxacin + ornidazole + zinc bisglycinate
72 metronidazole + norfloxacin
73 amoxicillin + bromhexine
74 ciprofloxacin + fluticasone + clotrimazole + neomycin is
75 metronidazole + tetracycline
76 cephalexin + neomycin + prednisolone
77 azithromycin + ambroxol
78 cilnidipine + metoprolol succinate + metoprolol tartrate
79 l-arginine + sildenafil
80 atorvastatin + vitamin d3 + folic acid + vitamin b12 + pyridoxine
81 metformin + atorvastatin
82 clindamycin + telmisartan
83 olmesartan + hydrochlorothiazide + chlorthalidone
84 l-5-methyltetrahydrofolate calcium + escitalopram
85 pholcodine + promethazine
86 paracetamol + promethazine
87 betahistine + ginkgo biloba extract + vinpocetine + piracetam
88 cetirizine + diethyl carbamazine
89 doxylamine + pyridoxine + mefenamic acid + paracetamol
90 drotaverine + clidinium + chlordiazepoxide
91 imipramine + diazepam
92 flupentixol + escitalopram
93 paracetamol + prochloperazine
94 gabapentin + mecobalamin + pyridoxine + thiamine
95 imipramine + chlordiazepoxide + trifluoperazine + trihexyphenidyl
96 chlorpromazine + trihexyphenidyl
97 ursodeoxycholic acid + silymarin
98 metformin 1000/1000/500/500mg + pioglitazone 7.5/7.5/7.5/7.5mg + glimepiride
99 gliclazide 80 mg + metformin 325 mg
100 voglibose+ metformin + chromium picolinate
101 pioglitazone 7.5/7.5mg + metformin 500/1000mg
102 glimepiride 1mg/2mg/3mg + pioglitazone 15mg/15mg/15mg + metformin 1000mg/1000mg/1000mg
103 glimepiride 1mg/2mg+ pioglitazone 15mg/15mg + metformin 850mg/850mg
104 metformin 850mg + pioglitazone 7.5 mg + glimepiride 2mg
105 metformin 850mg + pioglitazone 7.5 mg + glimepiride 1mg
106 metformin 500mg/500mg+gliclazide sr 30mg/60mg + pioglitazone 7.5mg/7.5mg
107 voglibose + pioglitazone + metformin
108 metformin + bromocriptine
109 metformin + glimepiride + methylcobalamin
110 pioglitazone 30 mg + metformin 500 mg
111 glimepiride + pioglitazone + metformin
112 glipizide 2.5mg + metformin 400 mg
113 pioglitazone 15mg + metformin 850 mg
114 metformin er + gliclazide Mr. + voglibose
115 chromium polynicotinate + metformin
116 metformin + gliclazide + piogllitazone + chromium polynicotinate
117 metformin + gliclazide + chromium polynicotinate
118 glibenclamide + metformin (sr)+ pioglitazone
119 metformin (sustainded release) 500mg + pioglitazone 15 mg + glimepiride 3mg
120 metformin (sr) 500mg + pioglitazone 5mg
121 chloramphenicol + beclomethasone + clomitrimazole + lignocaine
122 of clotrimazole + ofloxaxin + lignocaine + glycerine and propylene glycol
123 chloramphennicol + lignocaine + betamethasone + clotrimazole + ofloxacin + antipyrine
124 ofloxacin + clotrimazole + betamethasone + lignocaine
125 gentamicin sulphate + clotrimazole + betamethasone + lignocaine
126 clotrimazole + beclomethasone + ofloxacin + lignocaine
127 becloemthasone + clotrimazole + chloramphenicol + gentamycin + lignocaine ear
128 flunarizine + paracetamole + domperidone
129 rabeprazole + zinc carnosine
130 magaldrate + famotidine + simethicone
131 cyproheptadine + thiamine
132 magaldrate + ranitidine + pancreatin + domperidone
133 ranitidine + magaldrate + simethicone
134 magaldrate + papain + fungul diastase + simethicone
135 rabeprazole + zinc + domperidone
136 famotidine + oxytacaine + magaldrate
137 ranitidine + domperidone + simethicone
138 alginic acid + sodium bicarbonate + dried aluminium hydroxide + magnesium hydroxide
139 clidinium + paracetamol + dicyclomine + activated dimethicone
140 furazolidone + metronidazole + loperamide
141 rabeprazole + diclofenac + paracetamol
142 ranitidine + magaldrate
143 norfloxacin+ metronidazole + zinc acetate
144 zinc carnosine + oxetacaine
145 oxetacaine + magaldrate + famotidine
146 pantoprazole (as enteric coated tablet) + zinc carnosine (as film coated tablets)
147 zinc carnosine + magnesium hydroxide + dried aluminium hydroxide + simethicone
148 zinc carnosine + sucralfate
149 mebeverine &amp; inner hpmc capsule (streptococcus faecalis + clostridium butyricum + bacillus
Mesentricus + lactic acid bacillus)
150 clindamycin + clotrimazole + lactic acid bacillus

151 sildenafil + estradiol valerate
152 clomifene citrate + ubidecarenone + zinc + folic acid + methylcobalamin + pyridoxine + lycopene
+ selenium + levocarnitine tartrate + l-arginine
153 thyroxine + pyridoxine + folic acid
154 gentamycin + dexamethasone + chloramphenicol + tobramycin + ofloxacin
155 dextromethorphan + levocetirizine + phenylephrine + zinc
156 nimesulide + loratadine + phenylephrine + ambroxol
157 bromhexine + phenylephrine + chlorepheniramine maleate
158 dextromethorphan + bromhexine + guaiphenesin
159 paracetamol + loratadine + phenylephrine + dextromethorphan + caffeine
160 nimesulide + phenylephrine + caffeine + levocetirizine
161 azithromycin + acebrophylline
162 diphenhydramine + terpine + ammonium chloride + sodium chloride + menthol
163 nimesulide + paracetamol + cetirizine + phenylephrine
164 paracetamol + loratadine + dextromethophan + pseudoepheridine + caffeine
165 chlorpheniramine maleate + dextromethorphan + dextromethophan + guaiphenesin + ammonium
Chloride + menthol
166 chlorpheniramine maleate + ammonium chloride + sodium citrate
167 cetirizine + phenylephrine + paracetamol + zinc gluconate
168 ambroxol
+ guaiphenesin + ammonium chloride + phenylephrine + chlorpheniramine maleate + menthol
169 dextromethorphen + bromhexine + chlorpheniramine maleate + guaiphenesin
170 levocetirizine + ambroxol + phenylephrine + guaiphenesin
171 dextromethorphan + chlorpheniramine + chlorpheniramine maleate 
172 cetirizine + ambroxol + guaiphenesin + ammonium chloride + phenylephrine +
Menthol
173 hlorpheniramine + phenylephrine + caffeine
174 dextromethorphan + triprolidine + phenylephrine
175 dextromethorphan + phenylephrine + zinc gluconate + menthol
176 chlorpheniramine + codeine + sodium citrate + menthol syrup
177 enrofloxacin + bromhexin
178 bromhexine + dextromethorphan + phenylephrine + menthol
179 levofloxacin + bromhexine
180 levocetirizine + phenylephrine + ambroxol + guaiphenesin + paracetamol
181 cetirizine + dextromethorphan + phenylephrine + zinc gluconate + paracetamol + menthol
182 paracetamol + pseudoephedrine + dextromethorphan+cetirizine
183 diphenhydramine + guaiphenesin + ammonium chloride + bromhexine
184 chlorpheniramine + dextromethorphan + phenylephrine + paracetamol
185 dextromethorphen + promethazine
186 diethylcabamazine citrate + cetirizine + guaiphenesin
187 chlorpheniramine + phenylephrine + dextromethophan + menthol
188 ambroxol + terbutaline + dextromethorphan
189 dextromethorphan + chlorpheniramine + guaiphenesin
190 terbutaline + bromhexine + guaiphenesin + dextromethorphan
191 dextromethorphan + tripolidine + phenylephirine
192 paracetamol + dextromethorphan + chlorpheniramine
193 codeine + levocetirizine + menthol
194 dextromethorphan + ambroxol + guaifenesin + phenylephrine + chlorpheniramine
195 cetirizine + phenylephrine + dextromethorphan + menthol
196 roxithromycin + serratiopeptidase
197 paracetamol + phenylephrine + triprolidine
198 cetirizine + acetaminophen + dextromethorphan + phenyephrine + zinc gluconate 
199 diphenhydramine + guaifenesin + bromhexine + ammonium chloride + menthol
200 chlopheniramine maleate + codeine syrup
201 cetirizine + dextromethorphan + zinc gluconate + menthol
202 paracetamol + phenylephrine + desloratadine + zinc gluconate + ambroxol
203 levocetirizine + montelukast + acebrophylline
204 dextromethorphan + phenylephrine + ammonium chloride + menthol
205 acrivastine + paracetamol + caffeine + phenylephrine
206 naphazoline + carboxy methyl cellulose + menthol + camphor + phenylephrine
207 dextromethorphan + cetirizine
208 nimesulide + paracetamol + levocetirizine + phenylephrine + caffeine
209 terbutaline + ambroxol + guaiphenesin + zinc + menthol
210 dextromethorphan + phenylephrine + guaifenesin + triprolidine
211 ammomium chloride + bromhexine + dextromethorphan 
212 diethylcarbamazine + cetirizine + ambroxol
213 ethylmorphine + noscapine + chlorpheniramine
214 cetirizine + dextromethorphan + ambroxol
215 ambroxol + guaifenesin + phenylephrine + chlorpheniramine
216 paracetamol + phenylephrine + chlorpheniramine + zinc gluconate
217 dextromethorphan + phenylephrine + cetirizine + paracetamol + caffeine
218 dextromethophan + chlorpheniramine + guaifenesin + ammonium chloride
219 levocetirizine + dextromethorphan + zinc
220 paracetamol + phenylephrine + levocetirizine + caffeine
221 chlorphaniramine + ammonium chloride + sodium chloride
222 paracetamol + dextromethorphan + bromhexine + phenylephrine + diphenhydramine
223 salbutamol + bromhexine + guaiphenesin + menthol
224 cetirizine + dextromethorphan + bromhexine + guaifenesin
225 diethyl carbamazine + chlorpheniramine + guaifenesin
226 ketotifen + cetirizine
227 terbutaline + bromhexine + etofylline
228 ketotifen + theophylline
229 ambroxol + salbutamol + theophylline
230 cetririzine + nimesulide + phenylephrine
231 chlorpheniramine + phenylephrine + paracetamol + zink gluconate
232 acetaminophen + guaifenesin + dextromethorphan + chlorpheniramine
233 cetirizine + dextromethorphan + phenylephrine + tulsi
234 cetirizine + phenylephrine + paracetamol + ambroxol + caffeine
235 guaifenesin + dextromethorphan
236 levocetirizine + paracetamol + phenylephirine + caffeine
237 caffeine + paracetamol + phenylephrine + chlorpheniramine
238 levocetirizine + paracetamol + phenylephirine + caffeine
239 caffeine + paracetamol + phenylephrine + chlorpheniramine
240 ketotifen + levocetrizine
241 paracetamol + levocetirizine + phenylephirine + zink gluconate
242 paracetamol + phenylephrine + triprolidine + caffeine
243 caffeine + paracetamol + phenylephrine + cetirizine
244 caffeine + paracetamol + chlorpheniramine
245 ammonium chloride + dextromethorphan + cetirizine + menthol
246 dextromethorphan + paracetamol + cetirizine + phenylephrine
247 chlorpheniramine + terpin + antimony potassium tartrate + ammonium chloride + sodium
Citrate + menthol
248 terbutaline + etofylline + ambroxol
249 paracetamol + codeine + chlorpheniramine
250 paracetamol+pseudoephedrine+certirizine+caffeine
251 chlorpheniramine+ammonium chloride + menthol
252 n-acetyl cysteine + ambroxol + phenylephrine + levocertirizine
253 dextromethorphan + phenylephrine + tripolidine + menthol
254 salbutamol + certirizine + ambroxol
255 dextromethorphan + phenylephrine + bromhexine + guaifenesin + chlorpheniramine
256 nimesulide + certirizine + phenylephrine
257 naphazoline + chlorpheniramine + zinc sulphate + boric acid + sodium chloride + chlorobutol
258 paracetamol + bromhexine + phenylephrine + chlorpheniramine + guaifenesin
259 salbutamol + bromhexine
260 dextromethorphan + phenylephrine + guaifenesin + certirizine + acetaminophen
261 guaifenesin + bromhexine + chlorpheniramine + paracetamo
262 chlorpheniramine + ammonium chloride + chloroform + menthol
263 salbutamol + choline theophylinate + ambroxol
264 chlorpheniramine + codeine phosphate + menthol syrup
265 pseudoephedrine + bromhexine
266 certirizine + phenylephrine + paracetamol + caffeine + nimesulide
267 dextromethorphan + cetirizine + guaifenesin + ammonium chloride
268 dextromethorphan + cetirizine + guaifenesin + ammonium chloride
269 ambroxol + salbutamol + choline theophyllinate + menthol
270 paracetamol + chlorpheniramine + ambroxol + guaifenesin + phenylephrine
271 chlorpheniramine + vasaka + tolubalsm + ammonium chloride + sodium citrate + menthol
272 bromhexine + cetrizine + phenylephrine ip+guaifenesin + menthol
273 dextromethorphan + ambroxol + ammonium chloride + chlorpheniramine + menthol
274 dextromethorphan + phenylephrine + cetirizine + zinc + menthol
275 terbutaline + n-acetyl l-cysteine + guaifenesin
276 calcium gluconate + levocetirizine
277 paracetamol + levocetirizine + pseudoephedrine
278 salbutamol + choline theophylinate + carbocisteine
279 chlorpheniramine + vitamin c
280 calcium gluconate + chlorpheniramine + vitamin c
281 chlorpheniramine + paracetamol + pseudoephedrine + caffeine
282 guaifenesin + bromhexine + chlorpheniramine + phenylephrine + paracetamol + serratiopeptidase
(as enteric coated granules) 10000 sp units
283 paracetamol + pheniramine
284 betamethasone + fusidic acid + gentamycin + tolnaftate + lodochlorhydroxyquinoline (ichq
285 clobetasol + ofloxacin + miconazole + zinc sulphate
286 clobetasole + gentamicin + miconazole + zinc sulphate
287 levocetirizine + ambroxol + phenylephrine + paracetamol
288 permethrin + cetrimide + menthol
289 beclomethasone + clotimazole + neomycin + lodochlorohydroxyquinone
290 neomycin + doxycycline
291 ciprofloxacin + fluocinolone + clotrimazole + neomycin + chlorocresol
292 clobetasol + ofloxacin + ketoconazol + zinc sulphate
293 betamethasone + gentamicin + tolnaftate + lodochlorhydroxyquinoline
294 clobetasol + gentamicin + tolnaftate + lodochlorhydroxyquinone + ketoconazole
295 allantoin + dimethieone + urea + propylene + glycerin + liquid paraffin
296 acriflavine + thymol + cetrimide
297 betamethasone + neomycin + tolnaftate + lodochlorohydroxyquinoline + cholorocresol
298 clobetasol + neomycin + miconazole + clotrimazole
299 ketoconazole + tea tree oil + allantion + zinc oxide + aloe vera + jojoba oil +
Lavander oil + soa noodels
300 clobetasol propionate + ofloxacin + ornidazole + terbinafine
301 clobetasol + neomycin + miconazole + zinc sulphate
302 beclomethasone diproprionate + neomycin + tolnaftate + lodochlorhydroxyquinoline +
Chlorocresol
303 betamethasone + gentamycin + zinc sulphate + clotrimoazole + chlorocresol
304 borax + boric acid + naphazoline + menthol + camphor + methyl hydroxy benzoate
305 bromhexine + dextromethorphan
306 dextromethophan + chlopheniramine + bromhexine
307 menthol + anesthetic ether
308 dextrometharphan + chlopheniramine + ammonium + sodium citrate + menthol
309 ergotamine tartrate + belladona dry extarct+caffeine + paracetamol
310 phenytoin + phenobarbitone
311 gliclazide 40mg + metformin 400mg
312 paracetamol + ambroxol + phenylephrine + chlorpheniramine
313 oflaxacin + ornidazole suspension
314 albuterol + etofylline + bromhexine + menthol
315 albuterol + bromhexine + theophylline
316 salbutamol+hydroxyethyltheophylline (etofylline) + bromhexine
317 paracetamol+phenylephrine+levocetirizine+sodium citrate
318 paracetamol + propyphenazone + caffeine
319 guaifenesin + diphenhydramine + bromhexine + phenylephrine
320 dried alumnium hydroxie gel + prophantheline + diazepam
321 bromhenxine + phenylephrine + chlorpheniramine + paracetamol
322 beclomethasone + clotrimazole + gentamicin + lodochlorhydroxyquinoline
323 telmisartan + metformin
324 ammonium citrate + vitamin b 12 + folic acid + zinc sulphate
325 levothyroxine + phyridoxine + nicotinamide
326 benfotiamine + metformin
327 thyroid + thiamine + riboflavin + phyridoxine + calcium pantothenate + tocopheryl acetate +
Nicotinamide
328 ascorbic acid + manadione sodium bisulphate + rutin + dibasic calcium phosphate +
Adrenochrome mono semicarbazone
329 phenylephrine + chlorpheniramine + paracetamol + bromhexine + caffeine
330 clotrimazole + beclomethasone + lignocaine + ofloxacin + acetic aicd + sodium methyl paraben +
Propyl paraben

Avoid this combinations and be safe.

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Table of Content

About Montemac Plus 10 Mg/10 Mg Tablet
When is Montemac Plus 10 Mg/10 Mg Tablet prescribed?
What are the side effects of Montemac Plus 10 Mg/10 Mg Tablet?
Key highlights of Montemac Plus 10 Mg/10 Mg Tablet
What are the substitutes for Montemac Plus 10 Mg/10 Mg Tablet?
What are the interactions for Montemac Plus 10 Mg/10 Mg Tablet?