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Adolescent Problems Treatment
Limping Child Treatment
Management of New Born Care
Treatment of Newborn Jaundice
Treatment of Thyroid Disease in Children
Thyroid Disorder Treatment
Thyroid Problems Treatment
Adolescent Disorders Treatment
Treatment of Child and Adolescent Problems
Treatment of Childhood Diabetes
Cleft Lip Treatment
Management of Postnatal Care
Child Growth Management
Treatment of Childhood Infections
Management of Childhood Nutrition
Congenital Ear Problem Treatment
Quad Screening Treatment
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Parents nowadays give young children coke or Pepsi to drink. I was shocked to see a mother giving coke in a feeding bottle to a baby. Can anybody please provide the harmful effects of these drinks so that people don't make these mistakes. Thanks.
I have 23 month old child he catch two much cold and running nose and he is lethargic. I want to carry on with Ayurvedic medicine pls help me out. The problem is he gets running cold every 15 days we give him regularly tulsi water but still problem come again and again.
My sister's baby's age 41 days. His vaccination (PENTAVALENT +OPV1) is on 06.04. 2016. When can he be given ROTAVIRUS 1 Vaccine?
Hello sir My question is mera ladka hai uski age 6 th month hai. Wo kal sham se abi tak 7-8 baat pooty ki hai. Sir ye kia karan hai or iski medicine kya hai .
My 2 years old son has throat infection and white spot on his tonsils in every 20 to 30 days.After taking medicines including antibiotics he recover in 5 to 7 days and in some times in 10 days . It happens again and again. His doctor says its allergy and last in 5 to 7 years . In those days he eats very little and some times he drinks only water. Is there any solution?
I am 33 years. I have given birth to a girl baby on december 30th with normal delivery. Still i am not able to feed my daughter with breast milk. I have tried all the steps to increase molk supply but not able to produce milk. Could anyone help me with this?
How scared are you of root canals? what if we tell you. You no longer need to be?
Root canals are easily one of the most dreaded treatments in the world. I have seen patients compare the anxiety they feel before a root canal to things like open heart surgery and labour. The last thing you want to hear on a dental chair is the diagnosis that you need a root canal. The horror stories surrounding this dental treatment range from gruesome to excruciating.
This article is an attempt to dispel the mystery and pain associated with root canals and show you how far we have come from the horrors to the sophistication of the latest technology.
From painful to pleasing
What are root canals really?
In its simplest sense a root canal is a deep filling done by cleaning the infection from the third and innermost layer of the tooth which is made up of nerves and blood vessels.
Our tooth is made up of 3 layers the first 2 are hard and confined layers called enamel and dentin when decay affects these it is very slow to spread and easy to remove and fill within one short session.
The third layer may take 1-3 sessions to clean as the infection may have spread or collected in the supporting tooth structures.
Why are root canals considered painful?
The 3rd layer of our tooth is a nerve chamber containing soft nerves and blood vessels in communication with the rest of our body.
This is the place that communicates pain to our brain and this is why when decay or bacteria hit this soft deeper layer we experience sharp shooting pain.
Top 3 reasons why root canals used to be painful
Improper or inadequate anesthesia to numb the inflamed nerve
Mechanical instrumentation to manually pull out the nerve which we now dissolve and clean with automated machines
Lack of the right medications to use within the tooth.
What happens to root canal infections if left untreated?
If this pain is suppressed with medication and not treated it can lead to an infection spreading within the bone which may later lead to a swelling with pus etc.
If this infection is left within it can eat into the supporting bone and eventually infect or affect the adjacent teeth as well.
My breast milk in 6 months. Now my baby s 10 month old still he is not sitting properly. Just falling down within 30 secs. What to do?
A mother's prime focus always is how to ensure that her child is happy and comfortable, especially when the child is too small. One of the problems which bothers many mothers who are bringing up really small children, is diaper rash. Sensitive skin, tight diaper and prolonged contact with feces and urine etc are common reasons for diaper rash or irritant diaper dermatitis.
Here are few tips to avoid diaper rash and ensuring your child is always comfortable.
1. Careful with wash: if you are using a cloth diaper, be extra careful in washing such that there is no soap remaining in the cloth.
2. Clean well: when bathing your baby, make sure you clean the area covered by diaper with a mild soap and do not rub the area while drying, since the skin is sensitive.
3. Remove soiled diaper instantly: this is the most important. However busy you are, you need to aware of the times when the diaper/cloth has been soiled. And when soiled, the diaper has to be changed at the earliest. Do not keep your baby in the diaper for all hours of the day. It is good if they are out of the diaper for some time every day.
4. Keep the diaper loose. Also, ensure that you wash the cotton diapers with fabric softeners and try to avoid wipes altogether as they have strong chemicals in them.
5. Keep a tab on rashes: if your baby does get the rashes, check with the pediatrician at the earliest. Frequent topical applications of a bland protective barrier agent (zinc oxide paste) may suffice to prevent dermatitis. There are also chances of fungal infections. Antifungal creams should be used on advice of a doctor.
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Hi sir. My baby is 15 months old. He had a vaccine of typbar psv on 31 /1/16 from a good doctor in delhi And yesterday only I.e on 19th June one of the doctors in haryana applied him the 2nd doze. Of typbar psv and a doze of MMR 1$ ,he is having pain on his right leg and he vomited 2 times yesterday after eating some solid. He is having fever also.
Chronic hyperglycemia is captured by A1c but not by FPG (even when repeated twice).
Microangiopathic complications (retinopathy) are associated with A1c as strongly as with FPG.
A1c is better related to cardiovascular disease than FPG.
Fasting is not needed for A1c assessment.
No acute perturbations (e.G, stress, diet, exercise, smoking) affect A1c.
A1c has a greater preanalytical stability than blood glucose.
A1c has an analytical variability not inferior to blood glucose.
Standardization of A1c assay is not inferior to blood glucose assay.
Biological variability of A1C is lower than FPG and 2-h OGTT PG.
Individual susceptibility to protein glycation might be caught by A1c.
A1c can be used concomitantly for diagnosing and initiating diabetes monitoring
Natural history of T2DM in Asia
Diabetes is a global epidemic which is out of control, but worse in Asian countries.
It is a huge and growing problem and costs to the society are high and escalating.
Five countries from Asia figure in the top 10 and account for most cases of diabetes globally.
Asian countries share similar risk factors.
There is an association between economic growth and diabetes.
Rapid urbanization and modernization obesogenic environment i.E. Physical inactivity, psychosocial stress and abundance of food
Asians are prone to developing diabetes at a lower level of obesity.
Diabetes has the potential to negatively impact economy and may bankrupt healthcare systems.
Cost effective interventions in healthy living and diet decrease the burden of diabetes and save on healthcare costs and lost productivity.
There has been a dramatic rise in the number of diabetic population in Korea: economic growth, greater exposure to risk factors (lifestyle and diet), demographic changes (childhood obesity, aging population).
Hypertriglyceridemia: The most difficult lipid disorder to evaluate and treat
Hypertriglyceridemia is the most difficult lipid disorder to evaluate and treat. Hypertriglyceridemic disorder in adults is not a single gene. We do not know if TGs by themselves are an atherogenic risk or is it because of the company they keep.
The intra-individual biological variability (diurnal and monthly) of lipids make it more difficult to define hypertriglyceridemia.
TGs are inversely associated with HDL-C, if high HDL-C levels, almost always TGs are low.
Dietary treatment of severe hypertriglyceridemia: <5%, no alcohol, discontinue all TG-lowering drugs, monitor TG q 3 days until levels are below 1000, then restart treatment.
Fibrates do not reduce the CHD events in high risk patient groups. What impact hypertriglyceridemia has on CHD outcomes is not yet clear.
Lower fasting TG to less than 500 mg/dL; this will reduce the risk of pancreatitis.
Follow the current guideline recommendations to lower LDL-C.
The real value of Apo-B is in patients who do not have raised LDL-C (<100 mg/dL). In such patients it can be very informative and should be taken as an indicator of CVD risk.
Plasma apoB and the other cholesterol indexes are complementary rather than competitive indexes of atherosclerotic risk (Am J Cardiol. 2003 May 15;91(10):1173).
Baseline TGs are determinants of the response to bezafibrate (BIP trial).
Omega-3 fatty acids are beneficial in reducing CV risk (JELIS; Lancet 2007), especially in patients with high TG and low HDL-C (Atherosclerosis. 2008).
If fasting TG is >200 mg/dL and HDL-C <35 mg/dL, consider a fibrate or omega-3 fatty acid.